椎管内滑膜肉瘤误诊为混合型颈椎病一例

椎管内滑膜肉瘤误诊为混合型颈椎病一例李兴洲，张云岐，周洪伟患者男，６１岁。因右肩被碰撞后肩臂酸痛发胀、逐渐加重向下至双腿行走困难，胸腹部紧缩感、二便困难。体检：Ｃ５～６棘突右侧触、叩痛、颈后伸受限、右上肢肌肉萎缩，双侧Ｃ５以下浅感觉减弱、压顶试验（＋...     

VSD联合腓肠肌肌瓣治疗小腿复杂软组织缺损

目的探讨小腿复杂多发软组织缺损的修复方法。方法对地震伤后小腿多发软组织缺损造成骨外露,钢板外露的病患5例,经清创,腓肠肌肌瓣转移,封闭负压引流术（vacuum sealing drainage,VSD）覆盖创面7～10 d后行中厚皮片植皮治疗。结果移植肌瓣全部成活,创面一期愈合3～6个月,骨折愈合,伤口无感染。本组均获随访,随访时间5～10个月,平均7.5个月。膝踝关节功能无明显障碍,负重行走肢体无变形,无并发症。创面平均愈合时间为20d。结论 VSD技术联合腓肠肌肌瓣是治疗小腿复杂软组织缺损的有效方法。     

脊髓型颈椎病术后瘫痪及死亡原因分析

脊髓型颈椎病术后瘫痪及死亡原因分析李兴洲，王道新，王恩财本院自１９８５～１９９３年手术治疗脊髓型颈椎病３９７例，死亡２例（０．５％）、瘫痪５例（１．２６％）。本文重点对瘫痪及死亡发生的原因进行分析。１临床资料本组男５例，女２例，年龄５３～６４岁，平均...     

陈旧性骨结核误诊为软骨瘤三例

陈旧性骨结核误诊为软骨瘤三例李兴洲，石中令，龙海宇骨结核与骨肿瘤在病因、临床表现等方面截然不同，但骨结核在静止期尤其干酪样物钙化时，其影像表现与软骨瘤有相似之处，易于错误诊断。我们报告３例发生于特殊部位的陈旧性骨结核误为软骨瘤并分析误诊原因。例１女，...     

张力带内固定治疗尺骨鹰嘴骨折30例体会

临床资料1　一般资料从 1 995～ 2 0 0 0年我科对 3 0例尺骨鹰嘴骨折施行张力带内固定手术。男 2 1例 ,女 9例 ;年龄 1 8～ 60岁 ,平均 3 5岁。左 1 2例 ,右 1 8例。闭合性损伤 2 5例 ,开放性损伤 5例。骨折类型 :横形骨折 1 8例 ,斜行骨折 8例 ,粉碎骨折 4例。2　手术方法采用仰卧位臂丛麻醉 ,患肢外展 ,上气压止血带。取肘后正中切口 ,分离显露尺神经 ,保护尺神经以免术中损伤。清理骨折断端积血 ,少量剥离骨膜。将肘关节伸展约 1 2 0 0～ 1 3 0 0位置 ,放松肱三头肌 ,两手推动骨折断端使断端复位。准确复位后 ,用巾钳固定骨折端 ,于尺骨…     

大庆油田职业人群高尿酸血症危险因素分析*

摘 要：目的：比较家庭医生签约服务中签约居民与非签约居民对于新冠病毒疫苗接种满意度的差异,以此验证家庭医 生签约对新冠病毒疫苗接种工作的促进作用。方法：采用问卷调查所得数据建立数据库,主要使用个案控制匹配签约居民与 非签约居民,通过成组t检验进行统计分析。结果：签约居民组与未签约居民组各有260例匹配成功,匹配后两组协变量达 到均衡,匹配后签约居民组在 11个维度上的满意度均高于非签约居民组。结论：家庭医生签约有助于促进新冠病毒疫苗接 种工作,提升居民对社区卫生服务机构的利用和获得感。     

以《医学统计学》教学为例浅谈教案编写中的几点拙见

教案书写是教师的必修课,是教师对教学内容、教学方法的设计与实施,其重要性不言而喻.本文以医学统计学教学为例,提出教案编写中的几点做法,为高校教师实施切实可行的教学设计及教案编写提供一定的参考.     

克山病患者抗氧化能力测定

Objective To explore the relationship between myocardial damage and antioxidant capacity in vivo of Keshan disease patients and to analyze the possible pathogenesis. Methods In the period from 2005 to 2006, 41 chronic and latent Keshan disease cases were chosen as the case group from such serious endemic areas as Yongjin Village, Xinfa Village of Fuyu County, Fuan Village of Shangzhi County, Xinghuo Village of Wudalianchi County, 61 healthy people from the same area as internal controls, 48 healthy people from Xianglansan Village of Wangkui County, an un-endemic area, as external control. Fasting peripheral venous blood was collected from all the people. And blood selenium, glutathione peroxidase(GSH-Px) and suporoxide dismutase.(T-SOD) activities, malondialdehyde (MDA) levels were examined. Results Blood selenium level of the patient group [(34.80±13.30) μg/L], GSH-Px[(104.10±34.19)U/L]and T-SOD[(92.16±17.98)×103 U/L]actives were significantly lower than the internal control group [(41.24±13.57)μg/L, (118.57±25.49)U/L, (104.82±13.56)×103 U/L]and the external control group [(48.33±16.51)μg/L, (155.00±24.01)U/L, (108.48±12.73)×103 U/L], respectively, with a statistically significant difference(all P < 0.05). MDA level of the patient group[(7.12± 1.37)μmol/L]was higher than that in the internal control group[(5.36±1.18)μmol/L]and the external control group[(5.22±0.83)μmol/L]with a statistically significant differences(both P < 0.05). The blood selenium level, GSH-Px activity of internal control group was resoectively lower than that in the external control group, the differences being statistically significant(beth P < 0.05). Conclusions Selenium deficiency may lead to reduced antioxidant capacity and enhanced oxidative damage in Keshan disease patients in vivo. There may be a certain relationship between oxidative stress disorder and myocardial damage of Keshan disease.     

克山病患者抗氧化能力测定

Objective To explore the relationship between myocardial damage and antioxidant capacity in vivo of Keshan disease patients and to analyze the possible pathogenesis. Methods In the period from 2005 to 2006, 41 chronic and latent Keshan disease cases were chosen as the case group from such serious endemic areas as Yongjin Village, Xinfa Village of Fuyu County, Fuan Village of Shangzhi County, Xinghuo Village of Wudalianchi County, 61 healthy people from the same area as internal controls, 48 healthy people from Xianglansan Village of Wangkui County, an un-endemic area, as external control. Fasting peripheral venous blood was collected from all the people. And blood selenium, glutathione peroxidase(GSH-Px) and suporoxide dismutase.(T-SOD) activities, malondialdehyde (MDA) levels were examined. Results Blood selenium level of the patient group [(34.80±13.30) μg/L], GSH-Px[(104.10±34.19)U/L]and T-SOD[(92.16±17.98)×103 U/L]actives were significantly lower than the internal control group [(41.24±13.57)μg/L, (118.57±25.49)U/L, (104.82±13.56)×103 U/L]and the external control group [(48.33±16.51)μg/L, (155.00±24.01)U/L, (108.48±12.73)×103 U/L], respectively, with a statistically significant difference(all P < 0.05). MDA level of the patient group[(7.12± 1.37)μmol/L]was higher than that in the internal control group[(5.36±1.18)μmol/L]and the external control group[(5.22±0.83)μmol/L]with a statistically significant differences(both P < 0.05). The blood selenium level, GSH-Px activity of internal control group was resoectively lower than that in the external control group, the differences being statistically significant(beth P < 0.05). Conclusions Selenium deficiency may lead to reduced antioxidant capacity and enhanced oxidative damage in Keshan disease patients in vivo. There may be a certain relationship between oxidative stress disorder and myocardial damage of Keshan disease.