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Fetal exposure to 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin transactivates aryl hydrocarbon receptor‐responsive element III in the tyrosine hydroxylase immunoreactive neurons of the mouse midbrain
Authors:Takashi Tanida  Ken Tasaka  Eiichi Akahoshi  Mitsuko Ishihara‐Sugano  Michiko Saito  Shigehisa Kawata  Megumi Danjo  Junko Tokumoto  Youhei Mantani  Daichi Nagahara  Yoshiaki Tabuchi  Toshifumi Yokoyama  Hiroshi Kitagawa  Mitsuhiro Kawata  Nobuhiko Hoshi
Affiliation:1. Department of Anatomy and Neurobiology, Kyoto Prefectural University of Medicine, Kyoto, Japan;2. Department of Animal Science, Graduate School of Agricultural Science, Kobe University, Kobe, Japan;3. Frontier Research Laboratory, Corporate Research and Development Center, Toshiba Corporation, Kawasaki, Japan;4. Laboratory of Molecular and Cell Genetics, Graduate School of Biological Sciences, Nara Institute of Science and Technology, Nara, Japan;5. Laboratory of Molecular Oncology, Graduate School of Biological Sciences, Nara Institute of Science and Technology, Nara, Japan;6. Division of Molecular Genetics Research, Life Science Research Center, University of Toyama, Toyama, Japan
Abstract:
Keywords:2,3,7,8‐tetrachlorodibenzo‐p‐dioxin  aryl hydrocarbon receptor  aryl hydrocarbon receptor responsive element III  immunohistochemistry  tyrosine hydroxylase
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