伴基底节钙化的婴幼儿外伤后腔隙性脑梗塞

目的 探讨伴有基底节钙化的婴幼儿外伤后腔隙性脑梗塞的发病机理和治疗效果。方法 总结20例婴幼儿较轻微头颅外伤后发生脑梗塞的症状、影像学表现、治疗效果及预后。结果 婴幼儿外伤后脑梗塞均发生于一侧基底节区。临床表现为不同程度的一侧急性偏瘫、无意识改变及颅内高压症状。CT扫描发现双侧基底节区存在细小点状钙化，腔隙性脑梗塞灶毗邻钙化点，经治疗预后好，随访3个月全部康复。结论 婴幼儿外伤后腔隙性脑梗塞的发生与基底节钙化关系密切，但发病机理及基底节钙化的生理过程尚有待探究，本病诊断需与脑弓形虫病及巨细胞包涵体等相鉴别。

Pusttraumatic cerebral lacunar infarction in infants with basal ganglia calcifications

Objective To explore the pathologic mechanism and treatment of posttraumatic cerebral lacunar infarction in infants with basal ganglia calcifications. Methods The symptoms, radiation demonstrations, treatments and prognosis in 20 infants undergoing cerebral infarction following minor cerebral trauma were reviewed retrospectively. Results Posttraumatic cerebral infarctions all occurred in only one side of basal ganglia. It leads to acute hemiplegia in the patients, but no changes of consciousness and no signs of intracranial hypertension were noted. CT scan showed punctate calcification in bilateral basal ganglia and lacunar infarction just beside them. After treatment the infants recovered well within 3 months. Conclusions The occurrence of posttraumatic cerebral lacunar infarction is closely associated with basal ganglia calcification, but its mechanism is not clear. It needs to be differentiated from cerebral toxoplasmosis and cytomegalic inclusion disease.