Alcohol-dependent patients with neuroendocrine evidence for reduced dopamine d(2) receptor function have decreased platelet monoamine oxidase-B activity |
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| Authors: | Berggren U Fahlke C Balldin J |
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| Institution: | Institute of Clinical Neuroscience, Department of Psychiatry and Neurochemistry, Göteborg University, Sahlgrenska University Hospital/Mölndal, SE-431 80 Mölndal and
1 Department of Psychology, Göteborg University, Box 500, SE-405 30 Göteborg, Sweden |
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| Abstract: | This letter is a report of a re-evaluation of the results obtainedin an earlier study of ours (Balldin et al., 1994 ) of plateletmonoamine oxidase (MAO)-B activity in alcoholics with reduceddopamine (DA) D2 receptor function, as assessed by the growthhormone (GH) response to the D1/D2 agonist apomorphine (APO),which was published in this journal. The reason for the re-evaluationof the results in our study is a Letter to the Editors of thisjournal by Farren and Dinan (1996) reporting on platelet MAO-Bactivity in alcoholics with reduced DA D2 receptor function,as assessed by the GH response to the DA D2 receptor agonistbromocriptine. In the report by Farren and Dinan (1996), fourof eight alcoholics had no increase in GH above baseline, whereasall eight subjects in the control group had the expected GH. . . Full Text of this Article]FOOTNOTESREFERENCES |
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