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Resolution of Postural Orthostatic Tachycardia Syndrome After CT-Guided,Percutaneous T2 Ethanol Ablation for Hyperhidrosis
Authors:Malcolm Brock  Tae Hwan Chung  Sathvika Reddy Gaddam  Anjaneya Singh Kathait  Cecily Ober  Christos Georgiades
Affiliation:1.Department of Thoracic Surgery, Center for Sweat Disorders,Johns Hopkins University,Baltimore,USA;2.Physical Medicine and Rehabilitation,Johns Hopkins University,Baltimore,USA;3.Vascular & Interventional Radiology,Johns Hopkins University,Baltimore,USA;4.Department of Thoracic Surgery,Johns Hopkins University,Baltimore,USA
Abstract:Postural orthostatic tachycardia syndrome is characterized by orthostatic intolerance. Orthostasis (or other mild physical stress) triggers a cascade of inappropriate tachycardia, lightheadedness, palpitations, and often fainting. The underlying defect is sympathetic dysregulation of the heart, which receives its sympathetic tone from the cervical and upper thoracic sympathetic ganglia. Primary hyperhidrosis is also thought to be the result of sympathetic dysregulation. We present the case of a patient treated with CT-guided, percutaneous T2 EtOH sympatholysis for craniofacial hyperhidrosis. The patient also suffered from postural orthostatic tachycardia syndrome for many years and was unresponsive to treatment. Immediately after sympatholysis, the patient experienced resolution of both craniofacial hyperhidrosis and postural orthostatic tachycardia syndrome.
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