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基于多分析方法探讨近十年T细胞对类风湿关节炎影响的可视化分析
引用本文:苏小军,朱文菊,王欢,何倩,包强,郭影,可易弘,沈海丽,张志明.基于多分析方法探讨近十年T细胞对类风湿关节炎影响的可视化分析[J].中国现代医生,2024,62(12):1-8.
作者姓名:苏小军  朱文菊  王欢  何倩  包强  郭影  可易弘  沈海丽  张志明
作者单位:甘肃省中医院风湿骨病科,甘肃兰州 730050;甘肃中医药大学中西医结合学院,甘肃兰州 730030;兰州大学第二医院风湿免疫科,甘肃兰州 730030
基金项目:国家自然科学基金项目(82260880);甘肃省自然科学基金(22JR5RA626);甘肃省中医药管理局重点科研项目(GZKZ-2021-3)
摘    要:目的 通过对近十年T细胞对类风湿关节炎(rheumatoid arthritis,RA)影响的文献进行多软件可视化分析,总结该领域的研究热点及前沿发展。方法 在中国知网和Web of Science核心合集数据库检索2012—2022年关于T细胞对RA影响的中英文文献,运用CiteSpace、VOSviewer软件对其发文量、作者、关键词进行分析。结果 中文文献519篇,英文文献861篇,结果显示2020—2022年中文发文量缓慢增加,英文发文量整体趋势较为平稳。关键词分析显示,预测该领域的未来研究聚焦于T细胞在RA中的发病机制、RA的骨破坏机制、疾病活动度、氧化应激等方面。结论 T细胞对RA影响的研究关注度一直较高,研究价值大。但因国内外研究重点存在差异,团队间应良性互动、相互交流,以期揭示RA的内在机制,为靶向治疗提供理论依据。

关 键 词:类风湿关节炎  T细胞  CiteSpace  VOSviewer  可视化分析

Visual analysis of the impact of T cells on rheumatoid arthritis in the past decade based on multiple analysis methods
Abstract:Objective Through a multi-software visual analysis of the literature on the influence of T cells on rheumatoid arthritis (RA) in recent ten years, the research hotspot and frontier development in this field were summarized. Methods The Chinese and English literature on the influence of T cells on RA from 2012 to 2022 years was retrieved from CNKI and Web of Science database as the research object. CiteSpace and VOSviewer software were used to analyze the number of publications, authors and keywords. Results 519 articles in Chinese and 861 in English were retrieved. The results showed that the number of articles in Chinese increased slowly from 2020 to 2022 years, while the overall trend in English was stable. Keyword analysis shows that it is predicted that future research in this field will focus on the pathogenesis of T cells in RA, the mechanism of bone destruction in RA, disease activity, oxidative stress. Conclusion The influence of T cells on RA has attracted much attention in the past,present and future, and has great research value. However, due to the differences in research priorities at home and abroad, the teams should interact positively and communicate with each other to reveal the internal mechanism of RA and provide theoretical basis for targeted therapy.
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