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抗炎合剂通过TLR4/MyD88/NF-κB通路抑制脂多糖诱导的过表达高迁移率蛋白-1转染小鼠巨噬细胞系RAW264.7细胞炎症反应的机制研究
引用本文:闫国良,李淑芳,王馨璐,孙雪,汪海慧.抗炎合剂通过TLR4/MyD88/NF-κB通路抑制脂多糖诱导的过表达高迁移率蛋白-1转染小鼠巨噬细胞系RAW264.7细胞炎症反应的机制研究[J].河北中医,2020,42(9).
作者姓名:闫国良  李淑芳  王馨璐  孙雪  汪海慧
作者单位:上海中医药大学附属市中医医院急诊科,上海 200070,上海中医药大学附属市中医医院急诊科,上海 200070,上海中医药大学附属市中医医院急诊科,上海 200070,上海中医药大学附属市中医医院急诊科,上海 200070,上海中医药大学附属市中医医院急诊科,上海 200070
基金项目:上海中医药大学预算内项目
摘    要:

关 键 词:脓毒症  RAW264.7细胞  炎症因子  抗炎合剂  体外研究

Mechanism research of Kangyan mixture through TLR4/MyD88/NF-κB signaling pathway on inhibiting in-flammatory response of lipopolysaccharide -induced mouse macrophage cell line RAW264. 7 with overex-pression of high mobility group box 1 protein transfection
YAN Guoliang,LI Shufang,WANG Xinlu.Mechanism research of Kangyan mixture through TLR4/MyD88/NF-κB signaling pathway on inhibiting in-flammatory response of lipopolysaccharide -induced mouse macrophage cell line RAW264. 7 with overex-pression of high mobility group box 1 protein transfection[J].Hebei Journal of Traditional Chinese Medicine,2020,42(9).
Authors:YAN Guoliang  LI Shufang  WANG Xinlu
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