Bladder Autonomic Dysfunction after a Parietal Stroke |
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| Institution: | 2. Clinical Physiology Unit, Sakura Medical Center, Toho University, Sakura, Japan;2. Department of Epidemiology, Gilead Sciences, Inc., Foster City, California;3. Department of Neurology, Miller School of Medicine, University of Miami, Miami, Florida;4. Community Center, the Atahualpa Project, Atahualpa, Ecuador;2. Department of Neuroscience, Mayo Clinic, Jacksonville, Florida;3. Division of Hematology and Medical Oncology, Mayo Clinic, Jacksonville, Florida;4. Department of Laboratory Medicine & Pathology, Mayo Clinic, Jacksonville, Florida;5. Quality Management Services, Mayo Clinic, Jacksonville, Florida;2. St.Joseph''s Hospital and Medical Center, Phoenix, Arizona;3. OSF Healthcare System, Urbana, Illinois;4. University of Illinois, College of Medicine, Urbana, Illinois;2. Emergency Department, Jackson Memorial Hospital, Miami, FL;3. Department of Neurology, University of Maryland, Baltimore, MD |
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| Abstract: | We describe a case of a 57-year-old man who, immediately after a right parietal ischemic stroke, showed urodynamically determined bladder sensory decrement during filling and an underactive detrusor during voiding, both of which were ameliorated during the course of his treatment. The lower urinary tract symptom (LUTS) occurs in stroke in up to 60% of patients, when it involves the frontal and insular cortices. In addition, LUTS does occur in parietal stroke as seen in our patient, presumably by sensory deafferentiation within the brain that is relevant to the central regulation of the micturition reflex. |
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