妊娠高血压综合征胎盘组织结构分析

为探讨妊高征胎盘功能不良的形态学因素。应用体视学技术测定轻度（14例）、中度（10例）和重度（9例）妊高征胎盘，与14倒无妊娠并发症胎盘进行比较，结果显示：各组胎盘绒毛间腔、绒毛滋养细胞、绒毛毛细血管、绒毛间质体积百分含量无显著差异（P＞0．05）；胎盘梗死绒毛体积百分含量妊高征各组均高于对照组，差异有显著意义（P＜0．01）；重度妊高征组胎盘绒毛表面密度低于对照组，差异显著（P＜0.01）；中度和重度妊高征组胎盘绒毛毛细血管表面密度明显低于对照组（P＜0．01）。提示妊高征胎盘梗死绒毛增多、绒毛和绒毛毛细血管表面密度减少可导致胎盘功能不良。

HISTOLOGICAL STRUCTURE OF PLACENTA IN PREGNANCY-INDUCED HYPERTENSION

To evaluate the morphological parameters of placental dysfunction in the patients withpergnancy-induced hypertension (PIH ), 3 3 placentae with PIH (14,10 and 9 cases for mild, moderate and severe PIH resPectively)and 14 placentae with no-PIH were measured by stereologicaltechnique. Results: no significant difference was found in volume proportions among placental intervillous space, villous trophoblast, villous capillary and villous mesenchyme (P>0.05 ). Volumeproportions of placental infarct villi in PIH were significantly higher than those in no-PIH (P<0. 01). Placental villous surface density in severe PIH was significantly lower than that in no-PIH(P<0. 01). Surface density of placental capillary in moderate and severe PIH was significantlylower than that in no-PIH (P<0. 01). Indication: Increased placental infarct villi and decreasedsurface density of villi and villous capillary in the patients with PIH may lead to placental dysfunction,