Incomplete lacunar infarction (Type I b lacunes) |
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Authors: | G. A. Lammie F. Brannan J. M. Wardlaw |
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Affiliation: | (1) Neuropathology Laboratory, Department of Pathology, Edinburgh University, Alexander Donald Building, Western General Hospital, Crewe Road, Edinburgh EH4 2XU, UK Tel.: 44-131-537-1975, Fax: 44-131 537 1013, GB;(2) Department of Clinical Neurosciences, Edinburgh University, Western General Hospital, Crewe Road, Edinburgh EH4 2XU, UK, GB |
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Abstract: | The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia. Received: 21 October 1997 / Revised, accepted: 16 January 1998 |
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Keywords: | Lacune Infarction Basal ganglia Hypertension Embolus |
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