Involuntary movements associated with vitamin B12 deficiency |
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| Affiliation: | 1. Division of Radiology, Department of Pathophysiological Therapeutic Science, Faculty of Medicine, Tottori University, Japan;2. Division of Child Neurology, Institute of Neurological Sciences, Faculty of Medicine, Tottori University, Japan;1. Department of Cardiothoracic and Vascular Surgery, McGovern Medical School at The University of Texas Health Science Center at Houston, Houston, Texas;2. Memorial Hermann Heart & Vascular Institute, Texas Medical Center, Houston, Texas;3. Division of Vascular Surgery, Department of Surgery for Programmatic Development, Cedars-Sinai, Los Angeles, California;1. Cardiovascular Trials Unit, Central Manchester University Hospitals NHS Foundation Trust, Manchester, UK;2. Cardiovascular Research Group, Core Technology Facility, University of Manchester, Manchester, UK;3. Department of Clinical Biochemistry, Central Manchester University Hospitals NHS Foundation Trust, Manchester, UK;4. Department of Chemical Pathology, Trafford General Hospital, Manchester, UK;1. Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan;2. Department of Cardiac Surgery, The University of Tokyo Hospital, Tokyo, Japan;3. Department of General Medicine, Nagaoka Red Cross Hospital, Niigata, Japan;4. Department of Pathology, The University of Tokyo Hospital, Tokyo, Japan;5. Department of Internal Medicine, Nagaoka Red Cross Hospital, Niigata, Japan |
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| Abstract: | Involuntary movements are not a known feature of vitamin B12 (vB12) deficiency in adults, though they are a characteristic feature of vB12 deficiency in infants. This case report presents an adult patient with vB12 deficiency in whom, myoclonus-like muscular contractions appeared soon after the initiation of vB12 and disappeared after the first week of this therapy. To our knowledge, this is the first report of involuntary movements in an adult patient with vB12 deficiency. Although the mechanism remains unknown, involuntary movements similar to myoclonus should be considered as one of the extraordinary neurological manifestations of vB12 deficiency in adults. |
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