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Catalase induces the expression of inducible nitric oxide synthase through activation of NF-kappaB and PI3K signaling pathway in Raw 264.7 cells
Authors:Jang Byeong-Churl  Paik Ji-Hye  Kim Sang-Pyo  Bae Jae-Hoon  Mun Kyo-Chul  Song Dae-Kyu  Cho Chi-Heum  Shin Dong-Hoon  Kwon Taeg Kyu  Park Jong-Wook  Park Jong-Gu  Baek Won-Ki  Suh Min-Ho  Lee Soo Hwan  Baek Suk-Hwan  Lee In-Seon  Suh Seong-Il
Affiliation:Chronic Disease Research Center and Institute for Medical Science, Keimyung University School of Medicine, 194 DongSan-Dong, Jung-Gu, Daegu 700-712, Republic of Korea. Jangbc12@kmu.ac.kr
Abstract:
Keywords:AP-1, activator protein-1   AT, 3-amino-1,2,4-triazole   ERKs, extracellular-regulated protein kinases   GAPDH, glyceraldehyde 3-phosphate dehydrogenase   IFN-?, interferon-?   iNOS, inducible nitric oxide synthase   IRF, IFN-? responsive factor   JAK-2, Janus activated kinase-2   JNKs, c-Jun N-terminal kinases   NF-IL6, nuclear factor-interleukin 6   NF-κB, nuclear factor-kappa B   NO, nitric oxide   PI3K, phosphatidylinositol 3 kinase   PKC, protein kinase C   PTX, pertussis toxin   ROS, reactive oxygen species
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