| 共刺激分子与重症肌无力 |
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| 引用本文： | 黄芳,莫雪安. 共刺激分子与重症肌无力[J]. 神经损伤与功能重建, 2007, 2(2): 85-89 |
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| 作者姓名： | 黄芳 莫雪安 |
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| 作者单位： | 广西医科大学第一附属医院神经内科 南宁530021 |
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| 摘 要： | 在重症肌无力发病机制的研究中,共刺激分子的作用得到越来越多的重视。本文从CD28/B7、CT-LA-4/B7、CD40/CD40LI、COS/ICOSL、OX40/OX40L等共刺激分子在重症肌无力中的研究进展进行综述,对重症肌无力发病机制的认识提供一定帮助,为重症肌无力的病情观察和治疗提供新的线索。
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| 关 键 词： | 重症肌无力 共刺激分子 发病机制 |
| 文章编号： | 1001-117X（2007）02-0085-05 |
| 修稿时间： | 2007-02-09 |
Co-stimulatory Molecules and Myasthenia Gravis |
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| HUANG fang,MO Xue-an. Co-stimulatory Molecules and Myasthenia Gravis[J]. Neural Injury and Functional Reconstruction, 2007, 2(2): 85-89 |
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| Authors: | HUANG fang MO Xue-an |
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| Affiliation: | Department of neurology, the second affiliated hospital of guangxi medical university, Nanning 530021, China |
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| Abstract: | More and more attention was paid to the roles of co-stimulatory molecules in pathogenesis of myasthenia gravis(MG).This article reviews the advances on studying co-stimulatory molecules including CD28/B7,CTLA-4/B7,CD40/CD40L,ICOS/ICOSL,and OX40/OX40L in MG.The review may help the readers to master the newest knowledge of co-stimulatory molecules in the pathogenesis of MG and provide new clues for studying and treating this disease. |
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| Keywords: | myasthenia gravis co-stimulatory molecules pathogenesis |
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