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老年男性脑卒中后排尿异常尿动力学检查分析
引用本文:王亚仑,许予明,王焱,胡金华,芦山,张国贤,陈燕,李真珍,文建国.老年男性脑卒中后排尿异常尿动力学检查分析[J].临床泌尿外科杂志,2010,25(3):195-197,203.
作者姓名:王亚仑  许予明  王焱  胡金华  芦山  张国贤  陈燕  李真珍  文建国
作者单位:1. 郑州大学第一附属医院神经内科,郑州,450052
2. 郑州大学第一附属医院医学尿动力学中心;河南省高等学校临床医学重点学科开放实验室
摘    要:目的:探讨60岁以上老年男性脑卒中后排尿异常的尿动力学改变。方法:选择50例脑卒中后恢复期或后遗症期排尿异常老年男性患者,其中无BPH脑卒中22例,年龄(69.25±5.78)岁;有BPH脑卒中28例,年龄(71.49±7.65)岁;另选择20例无下尿路异常的正常老年男性(70.63±6.52)岁]作为对照组,分别进行尿动力学检查,并对各种尿动力学参数进行统计分析。结果:无BPH和有BPH脑卒中组比较,最大尿流率时逼尿肌压为(4.54±0.80)VS.(5.34±0.97)kPa(1cmH2O=0.098kPa),最大尿道闭合压为(5.45±0.55)VS.(6.99±1.05)kPa,剩余尿量为(9.0±3.4)VS.(12.9±5.3)ml,差异有统计学意义(P〈0.05)。与正常老年组比较,无BPH和有BPH脑卒中组逼尿肌反射亢进发生率均显著增加72.7%(16/22)VS.75%(21/28)VS.10.0%(2/20)],其最大膀胱容量明显减少(298.8±112.6)VS.(276.5±132.21)VS.(478.6±92.6)ml]。结论:脑卒中后排尿异常应进行尿动力学检查。有BPH脑卒中患者逼尿肌排尿压、尿道闭合压和剩余尿量变化较无BPH脑卒中患者更为明显,提示应同时治疗BPH。

关 键 词:脑卒中  排尿异常  尿动力学  前列腺增生

Urodynamic Evaluation of Bladder Functon in old Man with Abnormal Voiding After Stroke
Yalun WANG,Yuming XU,Yan WANG,Jinhua HU,Shan LU,Guoxian ZHANG,Yan CHEN,Zhenzhen LI,Jianguo WEN.Urodynamic Evaluation of Bladder Functon in old Man with Abnormal Voiding After Stroke[J].Journal of Clinical Urology,2010,25(3):195-197,203.
Authors:Yalun WANG  Yuming XU  Yan WANG  Jinhua HU  Shan LU  Guoxian ZHANG  Yan CHEN  Zhenzhen LI  Jianguo WEN
Institution:1Department of Neurology, First Affiliated Hospital of Zhengzhou University, Zhengzhou, 450052 ,China 2.ZMedical and Urodynamics Center, First Affiliated Hospital of Zhengzhou University ;3 Key Disciplines Clinical Laboratory of Colleges and Universities in Henan Province)
Abstract:Objective:To explore the urodynamic changes of the old man above 60 years with abnormal voiding after stroke. Methods:Urodynamic evaluation was performed in 50 old men with abnormal voiding after stroke including 28 cases(71.49±7.65 y) with benign prostatic hyperplasia(BPH) and 22 (69.25±5.78 y) with no BPH. Twenty old men(70.63±6.52 y) with normal voiding as control. The urodynamic parameters were compared between different groups. Results:There are significant differences between the stroke group without t3PH and those with BPH of maximal voiding detrusor pressure (Pdet. max. void)(4.54±0. 80 kPa vs. 5.34±0.97 kPa), maximal urethral closure pressure (Pmax. close, urethra) (5.45±0. 55 kPa vs. 6.99±1.05 kPa) and the residual (9.0 ±3.4 ml vs. 12.9±5.3 ml)(P〈0.05). Compared with normal controls, the patients without BPH and those with BPH showed a significant increase in detrusor hyperreflexia frequency72.7% (16/22) vs. 75%(21/28) vs. 10.0% (2/20)1] and their maximum bladder capacity decreased significantly(298.8± 112.6 ml vs. 276.5 ± 132.21 ml vs. 478.6±92.6 ml), P〈0.05. Conclusions:Abnormal voiding after stroke should carry out urodynamic examinations. The significant changes in urodynamic evaluation in eases with BPH after storke indicating the importance of treating BPH spontaneously during the stroke recovery in cases with abnormal voiding.
Keywords:stroke  abnormal voiding  urodynamics  benign prostatic hyperplasia
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