| 瘦素与冠心病 |
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| 引用本文： | 宋衍秋,夏大胜.瘦素与冠心病[J].中国心血管杂志,2005,10(2):144-146. |
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| 作者姓名： | 宋衍秋 夏大胜 |
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| 作者单位： | 天津市第一中心医院心内科,天津,300192 |
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| 摘 要： | 瘦素是肥胖基因编码的一种蛋白质产物,主要由白色脂肪组织分泌,通过与其受体结合发挥抑制食欲、减少能量摄入、增加能量消耗的生物学作用。大量临床研究表明冠心病患者常存在高瘦素血症,表现为瘦素抵抗。本文就瘦素在冠心病发病中的作用机制作一综述。
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| 关 键 词： | 瘦素 冠心病 机制 |
| 文章编号： | 1007-5410(2005)02-0144-03 |
| 修稿时间： | 2004年11月17 |
Leptin and coronary artery disease |
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| SONG Yan-qiu,XIA Da-sheng.Leptin and coronary artery disease[J].Chinese Journal of Cardiovascular Medicine,2005,10(2):144-146. |
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| Authors: | SONG Yan-qiu XIA Da-sheng |
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| Institution: | SONG Yan-qiu,XIA Da-sheng.Department of Cardiology,First Center Hospital,Tianjin 300192,China |
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| Abstract: | Leptin, the protein encoded by ob gene, is mainly produced by white adipose tissue. Leptin acts on a specific receptor located in the hypothalamus to decrease appetite and increase energy expenditure. Many studies indicated that in CHD patients, leptin levels were singnificantly higher and had leptin resistance. This article makes a review concerning the mechanism of leptin in the morbility of CHD. |
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| Keywords: | Leptin Coronary artery disease Mechanism |
| 本文献已被 CNKI 维普 万方数据 等数据库收录！ |
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