| 变异型心绞痛发病机制及其认识 |
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| 引用本文： | 曲茹虹,李璇,林宪如.变异型心绞痛发病机制及其认识[J].中华临床医师杂志(电子版),2014(8):1534-1537. |
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| 作者姓名： | 曲茹虹 李璇 林宪如 |
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| 作者单位： | 青岛市立医院心内科,山东省266011 |
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| 摘 要： | 变异型心绞痛是一种由冠状动脉痉挛所致的、心电图表现以ST段抬高为特征的特殊类型心绞痛。然而,冠状动脉痉挛是如何产生并持续作用目前尚未完全明确,可能与多种作用机制如冠状动脉内皮功能受损、自主神经功能紊乱、镁离子缺乏等因素有关。本文综述该类心绞痛发病机制的目前认识。
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| 关 键 词： | 心绞痛 变异型 冠状血管痉挛 内皮细胞 镁缺乏 发病机制 自主神经功能紊乱 |
Understanding of the pathogenesis of variant angina |
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| Qu Ruhong,Li Xuan,Lin Xianru.Understanding of the pathogenesis of variant angina[J].Chinese Journal of Clinicians(Electronic Version),2014(8):1534-1537. |
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| Authors: | Qu Ruhong Li Xuan Lin Xianru |
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| Institution: | (Department of Cardiology, Qingdao Municipal Hospital, Qingdao 266011, China) |
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| Abstract: | Variant angina is a special type of angina pectoris characterized by transient ST segment elevation in electrocardiogram and caused by coronary artery spasm. However, it is not yet entirely clear that how coronary spasm produces and maintains, which might involve a variety of mechanisms such as coronary artery endothelial impairment, autonomic nervous dysfunction and magnesium deficiency. In this article we reviewed the current understanding of the pathogenesis of variant angina pectoris. |
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| Keywords: | Angina pectoris variant Coronary vasospasm Endothelial cells Magnesium deficiency Pathogenesis Autonomic nervous dysfunction |
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