Complete reversal of tumoral calcinosis after subtotal parathyroidectomy in a hemodialysis patient |
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| Authors: | Younes Mohamed Belghali Safa Zrour-Hassen Saoussen Béjia Ismail Touzi Mongi Bergaoui Naceur |
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| Affiliation: | 1. Department of Biomedical Sciences, College of Veterinary Medicine, Cornell University, 240 Farrier Road, Ithaca, NY 14853, USA;2. Animal Health Diagnostic Center, Department of Population and Medicine, College of Veterinary Medicine, Cornell University, Ithaca, NY 14850, USA;3. United States Department of Agriculture, Agricultural Research Service, Beltsville Agricultural Research Center, Animal Parasitic Diseases Laboratory, Beltsville, MD 20705, USA;4. Department of Microbiology, The University of Tennessee, Knoxville, TN 37996. USA;1. Faculty of Veterinary Science, University of Sydney, New South Wales 2006, Australia;2. Department of Primary Industries, Parks, Water and Environment, Kings Meadows, Tasmania 7249, Australia |
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| Abstract: | IntroductionTumoral calcinosis is a rare complication of chronic hemodialysis whose mechanism is incompletely understood. The treatment is challenging and should target the main precipitating factors, most notably secondary hyperparathyroidism and calcium–phosphate (Ca×P) product elevation.Case reportIn this 41-year-old patient, tumoral calcinosis developed in the right hip and subacromial bursa of the right shoulder after 3 years of chronic hemodialysis. Laboratory tests showed hyperparathyroidism with Ca×P elevation. Subtotal parathyroidectomy was performed. Eight months later, there was no evidence of tumoral calcinosis at either of the previously affected sites.ConclusionThe effectiveness of parathyroidectomy in our patient underlies the key role played by secondary hyperparathyroidism in the pathogenesis of tumoral calcinosis complicating hemodialysis. |
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