| 炎症反应与缺血性脑卒中后出血转换 |
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| 引用本文： | 麦鸿成,陈丹霞,徐炳东,张玉生. 炎症反应与缺血性脑卒中后出血转换[J]. 海南医学, 2017, 28(22). DOI: 10.3969/j.issn.1003-6350.2017.22.029 |
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| 作者姓名： | 麦鸿成 陈丹霞 徐炳东 张玉生 |
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| 作者单位： | 暨南大学第一附属医院神经内科,广东 广州,510632 |
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| 基金项目： | 国家自然科学基金资助项目，广东省自然科学基金资助项目，广东省医学科研基金资助项目，广州市科技计划资助项目 |
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| 摘 要： | 缺血性脑卒中后出血转换(HT)是一种常见脑血管疾病并发症,较一般缺血性脑卒中有更高的致残率及病死率.目前文献报道,炎症反应是导致HT发生及加重的原因,其可通过参与氧化应激、激活基质金属蛋白酶、堆积炎症因子及血管异常生成等各方面起作用.本文就炎症反应参与HT发生的机制进行综述.
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| 关 键 词： | 炎症反应 缺血 脑卒中 出血转换 |
Inflammatory response and hemorrhagic transformation after cerebral ischemic stroke |
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| MAI Hong-cheng,CHEN Dan-xia,XU Bing-dong,ZHANG Yu-sheng. Inflammatory response and hemorrhagic transformation after cerebral ischemic stroke[J]. Hainan Medical Journal, 2017, 28(22). DOI: 10.3969/j.issn.1003-6350.2017.22.029 |
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| Authors: | MAI Hong-cheng CHEN Dan-xia XU Bing-dong ZHANG Yu-sheng |
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| Abstract: | Hemorrhagic transformation (HT) after cerebral ischemic stroke, with higher morbidity and mortality than normal cerebral ischemic stroke, is a common complication of cerebrovascular diseases. At present, literatures re-port that inflammatory response is a cause of HT aggravation by participating in oxidative stress, matrix metalloprotein-ases activation, inflammatory mediator accumulation and vessel abnormal growth. Herein, we review the mechanism of inflammatory response in HT. |
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| Keywords: | Inflammatory response Stroke Cerebral ischemic Hemorrhagic transformation |
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