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Calpain‐activated mTORC2/Akt pathway mediates airway smooth muscle remodelling in asthma
Authors:Y. Zeng  P.‐C. Cai  F. Liu  J. Yang  Y. Xia  Q. Zhang  L.‐J. Song  L.‐L. Zhou  F.‐Z. Li  Y.‐X. Lin  J. Fang  P. A. Greer  H.‐Z. Shi  W.‐L. Ma  Y. Su  H. Ye
Affiliation:1. Department of Pathophysiology, School of Basic Medicine, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, China;2. Department of Clinical Laboratory, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, China;3. Department of Respiratory and Critical Care Medicine, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei, China;4. Queen's University Cancer Research Institute, Kingston, ON, Canada;5. Department of Respiratory and Critical Care Medicine, Beijing Chaoyang Hospital, Capital Medical University, Beijing, China;6. Key Laboratory of Pulmonary Diseases, Ministry of Health of China, Wuhan, Hubei, China;7. Department of Pharmacology and Toxicology, Medical College of Georgia, Augusta University, Charlie Norwood Veterans Affairs Medical Center, Augusta, GA, USA
Abstract:
Keywords:airway smooth muscle cells  asthma  calpain  PI3K/Akt pathway  remodelling
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