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A common microdeletion affecting a hippocampus‐ and amygdala‐specific isoform of tryptophan hydroxylase 2 is not associated with affective disorders
Authors:Christian Hammer  Franziska Degenhardt  Lutz Priebe  Adrian M Stütz  Stefanie Heilmann  Sebastian M Waszak  Andreas Schlattl  Elisabeth Mangold  Per Hoffmann  Markus M Nöthen  Marcella Rietschel  Gudrun Rappold  Jan Korbel  Sven Cichon  Beate Niesler
Affiliation:1. Department of Human Molecular Genetics, University of Heidelberg, , Heidelberg, Germany;2. Institute of Human Genetics, University of Bonn, , Bonn, Germany;3. Department of Genomics, Life and Brain Center, University of Bonn, , Bonn, Germany;4. European Molecular Biology Laboratory, Genome Biology Research Unit, , Heidelberg, Germany;5. Institute of Neuroscience and Medicine (INM‐1), Research Center Juelich, , Juelich, Germany;6. Division of Medical Genetics, Department of Biomedicine, University of Basel, , Basel, Switzerland;7. German Center for Neurodegenerative Diseases (DZNE), , Bonn, Germany;8. Department of Genetic Epidemiology in Psychiatry, Central Institute of Mental Health, Medical Faculty Mannheim, University of Heidelberg, , Heidelberg, Germany
Abstract:
Keywords:5‐hydroxytryptamine (5‐HT)  bipolar affective disorder (BPAD)  copy number polymorphism (CNP)  deletion  major depressive disorder (MDD)  serotonin  tryptophan hydroxylase 2 (TPH2)
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