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Blockade of A2B adenosine receptor reduces left ventricular dysfunction and ventricular arrhythmias 1 week after myocardial infarction in the rat model
Affiliation:2. Gilead Sciences, Inc, Foster City, California;2. Institut d’Investigacions Biomèdiques August Pi I Sunyer (IDIBAPS), Barcelona, Spain;3. Department of Cardiology, Hospital Universitario Quirón-Madrid, Madrid, Spain;4. Department of Anatomy and Cell Biology, University of Extremadura, Badajoz, Spain;2. Department of Cardiology, Wuhan Asian Heart Hospital, Wuhan, People’s Republic of China;3. Department of Cardiac Surgery;4. Department of Pathology, Fuwai Hospital, National Center for Cardiovascular Diseases, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People’s Republic of China;2. Krannert Institute of Cardiology, Division of Cardiology, Department of Medicine, Indiana University School of Medicine, Indianapolis, Indiana;3. Dipartimento di Medicina Interna e Specialità Mediche, Policlinico Umberto I, University of Rome, Rome, Italy;5. Department of Neurology, Indiana University School of Medicine, Indianapolis, Indiana;4. Dipartimento di Medicina Clinica e Molecolare, Azienda Ospedaliera Sant’ Andrea, University of Rome, Rome, Italy;2. Department of Cardiology, Copenhagen University Hospital, Gentofte, Denmark;3. Departments of Medicine and Pharmacology, Vanderbilt University, Nashville, Tennessee;2. Department of Cardiothoracic Surgery, University of Pittsburgh Medical Center, Pittsburgh, Pennsylvania;3. Division of Cardiovascular Surgery, Hospital of the University of Pennsylvania, Philadelphia, Pennsylvania;5. Department of Cardiothoracic Surgery, John Muir Medical Center, Concord, California;4. Department of Cardiovascular and Thoracic Surgery, Aurora St. Luke’s Medical Center, Milwaukee, Wisconsin
Abstract:
Keywords:Myocardial infarction  Fibrosis  Optical mapping  Remodeling  Ventricular tachycardia
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