Ethylene Glycol Poisoning: Resolution of Cranial Nerve Deficit |
| |
| Affiliation: | 1. Centre for Perinatal Infection Research, The Children''s Hospital at Westmead, NSW, Australia;2. National Centre for Immunisation Research and Surveillance, The Children''s Hospital at Westmead, NSW, Australia;3. Sydney Medical School, The University of Sydney, NSW, Australia;4. Marie Bashir Institute for Infectious Diseases & Biosecurity, University of Sydney, NSW, Australia;5. Australian Paediatric Surveillance Unit, Sydney, NSW, Australia;6. The Children''s Hospital at Westmead, NSW, Australia |
| |
| Abstract: | Ethylene glycol poisoning is a major contributor to the development of idiopathic metabolic acidosis, which may lead to renal failure. Ethylene glycol poisoning should be among suspected differentials when assessing a seemingly intoxicated patient with hypocalcemia, anion gap acidosis, and nontoxic blood alcohol levels. Glycol intoxication may lead to delayed neurologic manifestations, which may lead to an inaccurate diagnosis. No clear clinical guidelines exist to recommend treatment for this late effect. This is a case of the complete resolution of facial nerve deficit secondary to glycol poisoning after the completion of a steroid therapy course on an inpatient psychiatric unit. |
| |
| Keywords: | calcium oxalate crystals cranial nerve deficit ethylene glycol fomepizole ingestion metabolic acidosis neurological sequelae of glycol intoxication palsy poisoning suicide toxicity |
| 本文献已被 ScienceDirect 等数据库收录！ |
|
