儿童过敏性紫癜消化道病变与幽门螺杆菌感染的相关性分析

目的分析儿童过敏性紫癜消化道病变与幽门螺杆菌感染的相关性。方法选取2018年1月至2019年6月我院收治的30例过敏性紫癜消化道病变患儿为分析组,另选择同时间段内的30名健康体检儿童为参照组。比较分析组和参照组的幽门螺杆菌感染情况;比较分析组幽门螺杆菌感染和非感染患儿的治疗效果及其他部位受累情况。结果分析组的幽门螺杆菌感染率明显高于参照组,差异具有统计学意义(P<0.05)。分析组幽门螺杆菌感染患儿的治疗总有效率明显低于非感染患儿,差异具有统计学意义(P<0.05);分析组幽门螺杆菌感染患儿其他部位受累总发生率明显高于非感染患儿,差异具有统计学意义(P<0.05)。结论幽门螺杆菌感染是导致过敏性紫癜患儿消化道病变的主要诱因之一,临床需针对过敏性紫癜患儿进行幽门螺杆菌检查,以明确病因,提高治疗效果。

Analysis of the relationship between alimentary canal diseases and Helicobacter pylori infection in children with anaphylactoid purpura

Objective To analyze the relationship between alimentary canal diseases and Helicobacter pylori infection in children with anaphylactoid purpura.Methods A total of 30 children with anaphylactoid purpura and alimentary canal diseases admitted in our hospital from January 2018 to June 2019 were selected as analysis group,and 30 healthy children in the same period were selected as reference group.The Helicobacter pylori infection were compared between the analysis group and the reference group;the therapeutic effects and other parts involvement of Helicobacter pylori infection children and non-infected children in the analysis group were compared.Results The infection rate of Helicobacter pylori in the analysis group was significantly higher than that in the reference group,and the difference was statistically significant(P<0.05).In the analysis group,the total therapeutic effect rate of Helicobacter pylori infection children was significantly lower than that of non-infected children,and the difference was statistically significant(P<0.05);in the analysis group,the total incidence of other parts involvement of Helicobacter pylori infection children was significantly higher than that of non infected children,and the difference was statistically significant(P<0.05).Conclusion Helicobacter pylori infection is one of the main causes of alimentary canal diseases in children with anaphylactoid purpura.It is necessary to carry out Helicobacter pylori test in children with anaphylactoid purpura,so as to clarify the cause the disease and improve the therapeutic effect.