应用大脑皮质体觉诱发反应地图验证循经感传的形成机制

The study is to observe the variation of functional motility in cortical somatosensory area I (SI) during propagated sensation and imitating sensation conduction along meridians with cortical somatosensory evoked potential(CSEP) topograpic map. The observation has been done on 42 volunteers and the results show that in those whose signs of propagated sensation along meiridians (PSM) were obvious when the sensation along the Gall Bladder Meiridian(GBM) passed to head and face, a red high potential signal appeared in the lower limbs representing area, which is near the middle line of cortical somatosensory evoked potential topograpic map, and a red high potential signal, jumping over the upper limbs representing area, also appeared in the face representing area, which is at the external part of cortical somatosensory evoked potential topograpic map, while in those whose PSM was not reported only a red high potential signal appeared in the lower limbs representing area. When Hegu (LI 4) was stimulated in those without PSM, usually an obvious evoked response appeared only in the upper limbs representing area. However, when Hegu was stimulted in those with PSM, the response area was larger in the upper limbs representing area and extending to face representing area. Mechanical compression can block PSM, and corresponding change will show in CSEP topographic map. This provides compelling evidence for the hypothesis Correspondence to: Professor Jin-sen Xu, Fujian Academy of TCM, Fuzhou 350003, China. Tel: (+86)591-8357-0748, Fax: (+86)591-8357-0007, Email: xujinsenjls@163.com that peripheral driver stimulation were the key element of producing PSM.

Formation mechanism of propagated sensation along the meridians, as verified by cortical somatosensory-evoked potential topographic maps

The peripheral driver stimulating theory states that as a driver passes along a certain meridian during acupuncture; the driver provokes nerve sense devices along the meridian, resulting in the nerve impulse entering the central nervous system. Accordingly, volunteers have reported propagated sensations along the meridians (PSM). The present study was designed to utilize a cortical somatosensory-evoked potential (CSEP) topographic map for determining whether stimulation expansion occurs in somatosensory area I when sensation was provoked in individuals with obvious PSM. The sensation was blocked by mechanical compression, and the sensation was imitated in individuals without PSM. Results revealed a red, high-potential signal in the representative area of the lower limbs in individuals with obvious PSM symptoms when the Gall Bladder Meridian (GBM) sensation passed to the head and face. This representative area was near the middle line of the CSEP topographic map, and a red, high-potential signal, which jumps over the representative area of the upper limbs, also appeared in the representative face area, which was at the external region of the CSEP topographic map. However, in individuals exhibiting no PSM, only a red high-potential signal appeared in the representative lower limb area. When Hegu (LI 4) was stimulated in individuals without PSM, an obvious evoked response appeared only in the representative upper limb area. However, when Hegu was stimulated in individuals exhibiting PSM, the response area was larger in the representative upper limb area and extended to the representative face area. When Guangming (GB 37) was stimulated in PSM individuals, the face representation response disappeared and was confined to a foot representation of the somatosensory area I when PSM was blocked by mechanical pressure. Results suggested that mechanical compression blocked PSM, and corresponding changes were exhibited in the CSEP topographic map. These results provide compelling evidence for the hypothesis that peripheral driver stimulation is the key element in producing PSM.