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NK-cell responses are biased towards CD16-mediated effector functions in chronic hepatitis B virus infection
Authors:Anita Schuch  Britta Franziska Zecher  Philipp Andreas Müller  Margareta P. Correia  Franziska Daul  Charlotte Rennert  Catrin Tauber  Karolin Schlitt  Tobias Boettler  Christoph Neumann-Haefelin  Hartmut Hengel  Hanspeter Pircher  Adelheid Cerwenka  Robert Thimme  Maike Hofmann
Affiliation:1. Department of Medicine II, University Hospital Freiburg — Faculty of Medicine, University of Freiburg, Hugstetter Straße 55, Freiburg 79106, Germany;2. Faculty of Biology, University of Freiburg, Schänzlestraße 1, Freiburg 79104, Germany;3. Innate Immunity Group, German Cancer Research Center, Im Neuenheimer Feld 280, 69120 Heidelberg, Germany;4. Institute of Virology, University Hospital Freiburg — Faculty of Medicine, University of Freiburg, Hermann-Herder-Straße 11, Freiburg 79104, Germany;5. Institute for Immunology, University Hospital Freiburg — Faculty of Medicine, University of Freiburg, Hermann-Herder-Straße 11, Freiburg 79104, Germany;6. Department of Immunobiochemistry, University of Heidelberg, Medical Faculty Mannheim, Ludolf-Krehl-Strasse 13-17, Mannheim 68167, Germany
Abstract:
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  • Keywords:NK cells  Chronic hepatitis B virus infection  Human cytomegalovirus  CD16  Memory-like  Adaptive
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