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肝硬化并发自发性腹膜炎腹水培养的病原菌及耐药性分析
引用本文:闫新. 肝硬化并发自发性腹膜炎腹水培养的病原菌及耐药性分析[J]. 中国实用医药, 2009, 4(21): 11-12
作者姓名:闫新
作者单位:河南省商丘市第一人民医院,467100
摘    要:目的探讨肝硬化并发自发性腹膜炎腹水培养的病原菌及耐药性并进行分析。方法本院2005年1月至2009年1月住院的肝硬化并发自发性细菌性腹膜炎242例患者腹水病原菌培养及药敏试验观察结果。结果42例腹水培养的阳性病原菌120株,阳性率为49.58%。G-杆菌占73.7%,G+球菌占15.9%。进行药敏试验,未出现耐药菌株7株(5.83%),产生耐药菌株113株(94.17%)。结论自发性腹膜炎早期诊断,及时足量地使用有效的抗生素治疗是改善晚期肝病患者预后的主要因素。用药应遵循早期、足量、联合、广谱、避免肝肾毒性的原则,正确合理和科学地使用抗菌药物,延缓细菌耐药性的产生。

关 键 词:肝硬化并发自发性腹膜炎  病原菌及耐药性  临床分析

Liver cirrhosis concurrent spontaneous peritonitis ascites raise's disease germ and the drug resistance analyzes
YAN Xin. Liver cirrhosis concurrent spontaneous peritonitis ascites raise's disease germ and the drug resistance analyzes[J]. China Practical Medical, 2009, 4(21): 11-12
Authors:YAN Xin
Affiliation:YAN Xin. (Henan Province Shangqiu First People's Hospital Shangqiu,Henan 467100, China)
Abstract:The discussion liver cirrhosis concurrent spontaneous peritonitis aseites raise's disease germ and the drug resistance and carry on the analysis. Method My courtyard is hospitalized January, 2005 liver cirrhosis concurrent spontaneity bacterial peritonitis 242 cases ascites disease germ raise which and the medicine sensitive test observation result to January, 2009. Results 42 example ascites raise' s masculine disease germ 120, the masculine gender rate is 49. 58%. The G- bacillus occupies the 73.7%, G + coccus to account for 15.9%. Enters takes a walk the sensitive experiment, has not appeared bears medicine strain 7 (5. 83% ), produces bears medicine strain 113 (94. 17% ). Conclusion The spontaneous peritonitis early diagnosis, the prompt sufficiency use effective antibiotic treatment is improves the later period liver disease patient prognosis the primary factor. The medication should follow the early time, the sufficiency, the union, Guang Pu, to avoid the hepatorenal toxic principle, correct reasonably and uses the antibiotics scientifically, delays the bacterium drug resistance production.
Keywords:Liver cirrhosis concurrent spontaneous peritonitis  Disease germ and drug resistance  Clinical analvsis
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