Quantitative aspects of L-type Ca2+ currents |
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| Authors: | Tuckwell Henry C |
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| Affiliation: | Max Planck Institute for Mathematics in the Sciences, Inselstr. 22, 04103 Leipzig, Germany |
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| Abstract: | |
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| Keywords: | AHC, auditory hair cells AM, atrial myocyte B, Brugada syndrome BAPTA, 12-bis (2-aminophenoxy) ethane-N, N, N&prime , N &prime -tetra-acetic acid BK, big conductance potassium channel Cai or [Ca2+]i, internal calcium ion concentration Cao or [Ca2+]o, external calcium ion concentration CDI, calcium-dependent inactivation CH, chromaffin CICR, calcium-induced calcium release CNS, central nervous system CORT, cortical DA, dopamine DCN, dorsal cochlear nucleus DRG, dorsal root ganglion dors, dorsal DRN, dorsal raphe nucleus EGTA, ethylene glycol (β-amino-ethyl ether)-N, N, N&prime , N &prime -tetra-acetic acid GABA, gamma-aminobutyric acid HEK, human embryonic kidney HVA, high-voltage activated IC, inferior colliculus KO, knockout LD, laterodorsal LVA, low-voltage activated Mag, magnocellular MR, medullary respiratory med, medial MID, midbrain MN, motoneuron N, neuron NRT, nucleus reticularis thalami PC, pituitary corticotroph PF, Purkinje fiber PM, pacemaker RF, renal failure SA, sino-atrial SE, serotonergic SH3-GK, src homology 3-guanylate kinase SK, small conductance potassium channel SM, skeletal muscle SMM, smooth muscle SN, substantia nigra SNc, substantia nigra pars compacta SON, supraoptic nucleus SS, disulphide bond SYMP, sympathetic TF, tetralogy of Fallot TR, thalamic relay VDI, voltage-dependent inactivation VM, ventricular myocyte VWA, Von Willebrand Factor A WT, wild-type |
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