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Modeling the impact of social problem-solving deficits on depressive vulnerability in the broader autism phenotype
Institution:1. Department of Psychology, Michigan State University, East Lansing, MI, United States;2. Division of Psychiatry and Behavioral Medicine, Michigan State University, East Lansing, MI, United States;3. Departments of Psychiatry and Ophthalmology, Rutgers University, Piscataway, NJ, United States;1. The Margaret and Wallace McCain Centre for Child, Youth & Family Mental Health and Campbell Family Mental Health Research Institute, Centre for Addiction and Mental Health, Toronto, ON, Canada;2. Centre for Brain and Mental Health and Department of Psychiatry, The Hospital for Sick Children, Toronto, ON, Canada;3. Department of Psychiatry, Faculty of Medicine, University of Toronto, Toronto, ON, Canada;4. Autism Research Centre, Department of Psychiatry, University of Cambridge, Cambridge, UK;5. Department of Psychiatry, National Taiwan University Hospital and College of Medicine, Taipei, Taiwan;6. Research Department of Clinical, Educational and Health Psychology, University College London, London, UK;1. Department of Psychology, University of South Florida, Department of Psychology, 4202 E. Fowler Avenue, PCD 4118G, Tampa, FL, 33620-7200, USA;2. Department of Pediatrics, University of South Florida, Department of Pediatrics, 12901 Bruce B. Downs Blvd, Tampa, FL, 33620-7200, USA;3. School of Aging Studies, University of South Florida, 4202 E. Fowler Avenue, MHC 1300, Tampa, FL 33620, USA;4. Rogers Behavioral Health—Tampa Bay, 2002 N. Lois Avenue, Suite 400, Tampa, FL 33607, USA;5. Department of Health Policy and Management, University of South Florida, Department of Health Policy and Management, 12901 Bruce B. Downs Blvd, Tampa, FL, 33620-7200, USA;6. Department of Psychiatry & Behavioral Neurosciences, University of South Florida, Department of Psychiatry and Behavioral Neuroscience, 12901 Bruce B. Downs Blvd, Tampa, FL, 33620-7200, USA;1. Department of Integrative Physiology, National Institute for Physiological Sciences, Okazaki, Aichi 444-8585, Japan;2. Graduate School of Engineering, Osaka University, Suita, Osaka 565-0871, Japan;3. Research Center for Child Mental Development, University of Fukui, Eiheiji, Fukui 910-1193, Japan;4. Department of Neuropsychiatry, Faculty of Medical Sciences, University of Fukui, Eiheiji, Fukui 910-1193, Japan;5. Biomedical Imaging Research Center, University of Fukui, Eiheiji, Fukui 910-1193, Japan;6. Department of Psychiatry, National Center of Neurology and Psychiatry Hospital, Kodaira, Tokyo 187-8551, Japan;7. Department of Child and Adolescent Mental Health, National Institute of Mental Health, National Center of Neurology and Psychiatry, Kodaira, Tokyo 187-8553, Japan;8. Research Center for Child Mental Development, Kanazawa University, Kanazawa, Ishikawa 920-8641, Japan;9. The University of Electro-Communications Graduate School of Informatics and Engineering Department of Informatics, Chofu, Tokyo 182-8585, Japan;10. Department of Cerebral Research, National Institute for Physiological Sciences, Okazaki, Aichi 444-8585, Japan;11. Department of Physiological Sciences, The Graduate University for Advanced Studies (Sokendai), Okazaki, Aichi 444-8585, Japan;1. University of Wisconsin, Department of Psychology, 800 Algoma Blvd., Clow F021, Oshkosh, WI 54901-8670, United States;2. University of Nevada, Department of Psychology and Program in Neuroscience, 1664 N. Virginia Ave. MS 296, Reno, NV 89557, United States
Abstract:The social communication and interaction deficits associated with the autism phenotype can have serious emotional consequences for individuals on the autism spectrum. This can be particularly true during young-adulthood, a period of increased social demands and expectations. The current study investigated the specific role of social problem-solving deficits as a mediator in the relationship between autism phenotype severity and depressive symptomology in young-adults. A sample of 230 university students (48% male) ranging in age from 18 to 30 (M = 21.30, SD = 2.48) were assessed on autism phenotype expression (Autism-Spectrum Quotient), social problem-solving ability (Social Problem-Solving Inventory, Revised) and depressive symptomology (Beck’s Depression Inventory). Results indicated that deficient social problem-solving skills account for a significant portion of the depressive symptomology associated with increased autism phenotype expression. Path model analysis output suggested that increased expression of the social components of the autism phenotype are associated with both ineffective social problem-solving styles and attitudes, while increased detail orientation discourages the use of an impulsive problem-solving style. The findings of this investigation provide preliminary evidence suggesting that programs designed to improve social problem-solving skills could be beneficial in the reduction of depressive vulnerability for young-adults on the autism spectrum.
Keywords:Autism-Spectrum Quotient (AQ)  Autism phenotype expression  Social problem solving  Depression  University students  Path model analysis
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