低钾性周期性瘫痪的临床研究

目的 进一步探讨低钾性周期性瘫痪病因、临床表现及预后等，为今后临床治疗低钾性周期性瘫痪提供帮助。方法 对141例低钾性周期性瘫痪患者进行观察，结合临床进行分析。结果 在本研究中我们发现一些与以往公认的低钾性周期性瘫痪理论不一致的地方：糖、胰岛素、醛固酮、肾上腺素等的水平也可改变钾的内平衡而促使瘫痪发作；周期性瘫痪患者可以出现腱反射活跃、正常等表现。结论 低钾性周期性瘫痪与血钾浓度、心电图的异常改变成正相关。临床上应结合辅助检查进一步明确诊断及时补钾到正常水平，以免耽误治疗。

Clinical observation on hypokalemic periodic paralysis

Objective To observe the clinical manifestations and cause of periodic paralysis and explore treatment method, Methods All the 141 periodic paralysis cases were clinically observed. Results It was observed that the levels of sugar, insulin, aldosterone , epinephrine ere could change the serum kaliemia. The results of ECG was normal in 86 cases, 71 cases showed normal deep tendon reflexes, 12 cases showed higher deep tendon reflexes and high level of serum CPK in 46 cases. Conclusion The serum hypokaliemia and ECG are good indicators for diagnosing the periodic paralysis. It is the key to maintain kalium t a normal level.