急性脑卒中并发低钠血症的临床意义及机制探讨

目的 探讨急性脑卒中并发低钠血症的机制及临床意义。方法 306例急性脑卒中患者入院后24 h内抽取外周静脉血5 ml,取血清1.5 ml,用全自动生化仪监测血清钠,治疗1周后复查。结果 24 h内有70例发生低钠血症,占22.9％,1周后发生低钠血症者66例,占21.6％,其中脑出血最多,其次蛛网膜下腔出血,脑梗塞最少(P<0.005)。结论 急性脑卒中并发低钠血症与卒中部位和性质有关。急性脑卒中患者并发低钠血症时预后不良。

The Mechanism and the Clinical Significance of Acute Cerebral Death Complicated Hyponatremia

Objective Discuss the mechanism and the clinical significance of acute cerebral death complicated hyponatremia. Methods After 306 patients of acute cerebral death be hospitalized within 24 hours,draw 5ml external venous blood and 1. 5ml serum. Research the serum sodium by full -automatic and reexamine after one week. Results There are 70 people who occur hyponatremia within 24 hours. It is 22. 9 percent. There are 66 people who occur hyponatremia within one week. It is 21. 6 percent. Among them,the cerebral hemorrhage patients are at most. Then,among the subarachnoid hemorrhage patients, the patients of cerebral infarction are at least. Conclusions Acute cerebral death complicated hyponatremia is related to the location of death and character. Acute cerebral death complicated hyponatremia patients have unfavourable prognosis.