乳牙深龋时冠部牙髓的病理变化和细菌分布

目的 从临床和实验研究两方面探讨深龋乳牙冠髓病理学状况及导致冠髓病变的原因。方法 随机选择Ｈｅ面深龋乳磨牙４５颗，行活髓切断术，光镜下观察其冠髓病理改变；用需氧、厌氧培养技术研究其髓腔内细胞；用扫描电镜观察窝洞底部牙本质小管中细菌的种类和分布特征。结果 ７７．８％的冠髓有不同程度的慢性炎症或慢性牙髓炎急性发作；９４．９％的髓腔中有需、厌氧菌混合感染；洞底牙本质小管中有球菌和杆菌侵入，球菌均位于小管

The study of histopathology and bacteriology of coronal pulp tissue in deciduous teeth with deep dentin caries

Objective To study the status of coronal pulp of deciduous teeth with deep caries and causes of pulpal histopathological changes Methods 45 deciduous molars with occlusal deep caries were selected at random The patients did not receive any antibiotic for one month The coronal pulpal histopathology was observed with optical microscopic technique The bacteria in pulp chamber were cultured by the ways of aerobic and anaerobic culture technique The dentinal tubular structures and bacteria within the tubules of cavity basal part dentin were studied with scanning electron microscopic technique Results Coronal pulp tissue of 77 8% of deciduous teeth with deep caries showed chronic inflammation to various degrees or chronic pulpitis with acute attack There were aerobic and anaerobic mixed infection in pulp chambers of 94 9% deciduous teeth with deep caries It was found that coccus and bacillus invaded into dentinal tubules of cavity basal part Coccus located inside tubules with intact tubular walls, while tubular walls were destroyed the bacillus was present Conclusion There are aerobic and anaerobic mixed infection in pulp chamber of deciduous teeth with deep caries The coronal pulp tissue shows chronic inflammation to various extent in over 2/3 deciduous teeth with deep caries Bacillus has stronger destructive power to dental tubules