从气虚络瘀探析支气管哮喘慢性持续期病机演变

支气管哮喘慢性持续期可出现气虚络瘀病机变化,肺脾肾气虚为本,血络瘀阻为标。认为本病气虚络瘀病机演变过程为久病耗气伤阴,进而导致气阴亏虚,子病及母,肺气亏虚,脾气亦不足,脾气虚则气血生化乏源,后天失养可致肾气亦不足。同时,脾气不足,水液运化失常而致水湿停聚,聚而为痰,亦可导致痰浊阻肺;肾气亏虚,纳气失常,可出现喘息气促。气虚久病入络,无力推动血液运行,血行迟缓而致血液瘀滞不行。

Pathogenesis Development of Bronchial Asthma in Chronic Duration from Qi Deficiency and Collateral Blood Stasis

Qi deficiency and collateral blood stasis were the main pathogeneses in chronic duration of bronchial asthma. Qi deficiency of the lung,spleen and kidney was the root while collateral blood stasis was the branch. The pathogenesis development of Qi deficiency and collateral blood stasis included :chronic illness consuming Qi and Yin caused Qi and Yin deficiency. Child disease involving mother organ,so lung deficiency involved spleen deficiency resulting in Qi and blood deficiency,further leading to kidney deficiency. Meanwhile,the spleen deficiency leading to water and dampness accumulation,eventually resulted in phlegm blocking lung collateral. Kidney Qi deficiency caused wheezing and shortness of breath. Chronic illness into the collaterals and deficient Qi was unable to promote blood circulation,resulting in blood stasis.