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Jane Leserman PhD Douglas A. Drossman MD Zhiming Li MD PhD 《Behavioral medicine (Washington, D.C.)》2013,39(3):141-142
Abstract Epidemiology and Health Policy Michel A. Ibrahim Rockville, Md., Aspen Publications, 1985. 197 pp., $29.95 Reviewd by Berton H. Kaplan, PhD Stress, Appraisal, and Coping Richard S. Lazarus and Susan Folkman New York, Springer, 1984. 464 pp. $19.95 Reviewd by David S. Strogatz, PhD 相似文献
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Govind K Makharia Chris J J Mulder Khean Lee Goh Vineet Ahuja Julio C Bai Carlo Catassi Peter H R Green Siddhartha Datta Gupta Knut E A Lundin Balakrishnan Siddartha Ramakrishna Ramakant Rawat Hanish Sharma Ajit Sood Chikako Watanabe Peter R Gibson World Gastroenterology Organization‐Asia Pacific Association of Gastroenterology Working Party on Celiac Disease 《Journal of gastroenterology and hepatology》2014,29(4):666-677
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Objective
The history of treatments for fistula-in-ano can be traced back to ancient times. Current treatment of transphincteric fistulae is controversial, with many options available. We reviewed the history of treatment using cutting setons and present our series of transphincteric fistulae in the light of the series in the literature.Design
Literature review and case series.Setting
Hospital based coloproctology serviceParticipants
140 consecutive patients presenting with fistula-in-ano were included.Main Outcome Measures
The literature pertaining to treatment of transphincteric fistula was reviewed, along with the outcome of various treatment methods for this condition. Data were collected for 140 consecutive patients presenting with fistula- in-ano were assessed for fistula healing, recurrence and complications.Results
A total of 140 consecutive patients with fistula-in-ano were identified, of which 111 were cryptoglandular (79.3%). Eighty-one of these 111 were transphincteric (73.0%). At a median follow-up of 35 months (range, 2–83 months), 70 transphincteric fistulae had healed (86.4%), 10 were still undergoing treatment (12.3%) and one patient was lost to follow-up prior to treatment (1.2%). Two patients in this group required a stoma (2.5%), six patients developed recurrence (7.4%); three ‘true’ recurrences (3.7%). One (1.2%) developed a chronic fissure. There were no reported cases of incontinence.Conclusions
The management of transphincteric fistula-in-ano is complex and controversial, for which no clear surgical procedure has gained acceptance as the gold standard. This study demonstrates that transphincteric fistulae can be successfully treated using cutting setons. A high healing rate (86.4%), low recurrence rate (7.4%) and a low complication rate (3.7%) are shown, which compares favourably with published rates over a long follow-up. 相似文献9.
Maria Saito‐Benz Helen Elizabeth Miller Mary Judith Berry 《Journal of paediatrics and child health》2015,51(12):1228-1231
A preterm neonate at 29‐week gestational age was born with intrauterine growth restriction, severe pancytopaenia and gross skeletal dysplasia. Antenatal screening bloods, TORCH/parvovirus tests and karyotype were unremarkable. Postnatally, he had normal microarray comparative genomic hybridization and serum B12/folate levels, and human immunodeficiency virus and cytomegalovirus polymerase chain reaction and antoimmune screening were negative. Targeted gene testing for Shwachman–Diamond syndrome (SDS) revealed the pathognomic mutation (c.183_184delTAinsCT). His postnatal clinical course was complicated by: (i) Ventilator dependency because of a combination of a pathologically compliant chest wall and preterm‐associated chronic lung disease. (ii) Progressive bone marrow failure, resulting in transfusion dependence and profound neutropenia associated with recurrent sepsis. (iii) Gastrointestinal failure and TPN dependency. (iv) Poor postnatal growth with weight/length/head circumference all <3rd centile. (v) Prognostication was complicated by the lack of published literature on the presentation of SDS in a preterm infant. However, because of inexorable progression of multiorgan failure, intensive care was withdrawn on day 54 of life. SDS is a rare autosomal recessive disorder characterised by haematological abnormalities, skeletal dysplasia and exocrine pancreatic dysfunction. Neonatal presentation is thought to be extremely rare. However, with the availability of genetic testing, it has now become clear that because of overlap in clinical presentation, term‐born infants with skeletal dysplasia and severe respiratory distress may initially be misdiagnosed as asphyxiating thoracic dystrophy. This case report highlights the complexities of preterm birth complicating clinical manifestations of SDS. 相似文献
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