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1例因房室传导阻滞(AVB)植入具有心室起搏管理(MVP)功能起搏器患者的心电图,当房室传导功能正常时,以AAIR模式工作。出现间歇或短暂AVB,提供心室备用起搏。出现持续性AVB,转换为DDDR模式工作。具有MVP功能的起搏器可降低累计心室起搏百分比。 相似文献
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Objective To investigate the mechanism of enhanced large conductance calciumactivated potassium channel currents (BK) in coronary smooth muscle cells (SMCs) by docosahexaenoic acid (DHA). Methods Coronary SMCs were isolated by enzyme digestion. Potassium channels in coronary SMCs were identified by applications of different potassium blockers. Effects of DHA and its metabolite 16,17-epoxydocosapentaenoic acid (16,17-EDP) on BK channels in the absence and presence of cytochrome P450 epoxygenase inhibitor SKF525A were studied by patch clamp in whole-cell configuration. Results BK channels were widely distributed in SMCs, and BK currents in normal SMCs accounted for (64.2±2.7)%of total potassium currents(n =20). DHA could activate BK channels, and its 50% effective concentration (EC50) was (0.23±0.03)μmol/L, however, the effect of DHA on BK channels was abolished after SMCs were incubated with cytochrome P450 epoxygenase inhibitor SKF525A. 16,17-EDP, a metabolite of DHA, could reproduce the effects of DHA on BK channels, and its EC50 was (19.7± 2.8) nmol/L.Conclusion DHA and metabolites can activate BK channels and dilate coronary arteries through activating cytochrome P450 epoxygenase pathway. 相似文献
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临时心脏起搏应用于严重缓慢性心律失常的紧急处理,安全有效,但也可能导致严重的并发症。本文报道1例高度房室传导阻滞患者,临时心脏起搏术后心电图、超声心动图、胸部CT提示临时起搏导线经心内膜垫缺损致左心室起搏,需重视临时心脏起搏并发症。 相似文献
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患者女性,75岁。贯序使用4个起搏系统。旷置电极导线使局部感染经抗生素及清创治疗难以控制,导致心脏装置相关感染性心内膜炎,外科直视手术彻底清除感染灶后治愈。再次植入起搏器时发现心肌组织在炎症、手术创伤后形成纤维化而使起搏阈值明显升高。 相似文献
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目的探讨CK-Mb/CK比值诊断AMI的价值。方法检测68例急性心肌梗死(AMI)患者血肌酸激酶同工酶Mb(CK-Mb),肌酸激酶(CK),肌红蛋白(Myo),肌钙蛋白I(cTnI),分析AMI后第1个24h内每6小时CK-Mb/CK比值;AMI6h内CK-Mb/CK〉10%,Myo,cTnI阳性率比较。结果AMI第一个6h内CK-Mb/CK显著高于之后的数小时,Myo,CK-Mb/CK〉10%及cTnI三者阳性率间无差别。结论CK-Mb/CK为一项早期诊断及鉴别诊断AMI的实用指标。 相似文献
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2型糖尿病患者真胰岛素和胰岛素原的改变及其意义 总被引:1,自引:0,他引:1
目的:探讨2型糖尿病(DM)患者、糖耐量减低(IGT)者血清真胰岛素(TI)、胰岛素原(PI)及免疫反应胰岛素(IRI)的变化及其意义。方法:测定42例糖耐量正常者(NGT)、31例IGT和91例新诊断的2型DM患者空腹及餐后1h、2h的TI、PI和IRI。并根据体重指数(BMI)分为肥胖晋组和非肥胖亚组。结果:(1)各组TI、PI和IRI在肥胖亚组均比非肥胖亚组高(P<0.01)。(2)2型DM肥胖和非肥胖亚组餐后1h的TI和IRI明显低于NGT的相应组(P<0.05),也低于2型DM餐后2h的相应组,TI和IRI释放曲线呈早期分泌相降低和高峰延迟。(3)空腹PI:NGT<IGT<2型DM组,各组之间差异有显著性(P<0.05)。(4)各组内肥胖亚组的空腹PI/IRI和非肥胖亚组相似(P>0.05),2型DM、IGT的空腹PI/IRI显著高于NGT(P<0.05)。结论:不同糖耐量的肥胖者均有高胰岛素血症和高胰岛素原血症。2型DM、IGT患者有高PI血症和不成比例的PI增高,后者是胰岛β细胞分泌功能缺陷的标志之一。 相似文献