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YUKAKO ISHIHARA MASAHIRO SEIKE ITARU YOKOUCHI HIROAKI SUDA SHUNICHIRO ISHITSUKA TADAYOSHI KAKEMURA MITSUHIRO YOSHIDA KAZUYA YOSHIMOTO YOSHIHIRO SAKAI CHIDORI OKUMURA YOSHIHISA SAIDA 《Digestive endoscopy》2003,15(3):240-242
A 55‐year‐old‐man had a laparoscopic resection of the sigmoid colon due to colon cancer with submucosal invasion. After the surgery he suffered ileus and had a laparotomy. Six months later he complained of frequent defecation. Colonoscopy confirmed a circular ulcer extending from the anal side of the anastomosis in the sigmoid colon to the mid rectum. Endoscopic ultrasound demonstrated thickening of all layers of the diseased colon and rectum. We diagnosed ischemic colitis. After intravenous drip infusion of prostaglandin, symptoms and colonic stricture gradually improved. Although abdominal angiography revealed a narrowing of the peripheral sigmoid branch of the inferior mesenteric artery, blood flow was unrestricted. Colonoscopy performed 84 days after discharge revealed an ulcer scar. 相似文献
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SHUNICHIRO SUMI M.D. SEIYO MARUYAMA M.D. YUHEI SHIGA M.D. SUNAO KODAMA M.D. KEI MIYOSHI M.D. HIDEAKI TOJOU M.D. YOSHIO YAMANOUCHI M.D. HIDENORI URATA M.D. 《Pacing and clinical electrophysiology : PACE》2010,33(6):e53-e56
The patient was a 57‐year‐old man with Brugada syndrome, who had been implanted with a implantable cardioverter defibrillator (ICD). The frequency of ventricular fibrillation (VF) started to increase about 4 years after ICD implantation, occurring, at worst, six times in one night. Immediately after starting oral administration of disopyramide, VF stopped occurring. He then discontinued taking disopyramide, but immediately after the discontinuation VF started occurring again, so he restarted taking disopyramide. Thereafter, VF completely stopped occurring. Findings observed in our case suggest that disopyramide could be added in our arsenal of medications for treating arrhythmic storms in patient with Brugada syndrome. (PACE 2010; 33:e53–e56) 相似文献
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SHUNICHIRO MIYOSHI M.D. TOSHTHISA MIYAZAKT M.D. MIKA ASANAGI M.D. KAZUNORI MORITANI M.D. SATOSHI OGAWA M.D. 《Journal of cardiovascular electrophysiology》1998,9(3):292-298
Epicardial and and Endocardial [K+]0 Rise and KATP Channels. Introduction: KATP channels are activated predominantly in the epicardium during regional ischemia. Therefore, the role of KATP channels in ischemia-induced rise of extracellular potassium concentration ([K+]o) might he greater in the epicardium. Methods and Results: In 18 anesthetized dogs, the left anterior descending coronary artery (LAD) was ligated, followed by injection of 23-μm latex heads into the occluded artery to interrupt collateral flow, by which accumulated [K+]o might wash out. Epicardial and endocardial [K+]o were measured during a 20-minute period of ischemia using a valinomycin membrane. The dogs were divided into three groups: 6 control dogs (CTRL); 7 dogs pretreated with intravenous glibenclamide (0.3 mg/kg [GLIB]), a blocker of KATP channels: and 5 dogs pretreated with intravenous nicorandil (0.2 to 0.25 mg/kg [NCR]), a KATP channel opener. Before LAD occlusion, there was no difference in [K+]o among the three groups. In the control group, epicardial and endocardial [K+]o were increased to a similar level as a function of time after occlusion (CTRL) at both layers. Ischemia-induced epicardial [K+]o rise was suppressed by GLIB (8.4 ± 0.4 vs 6.7 ± 0.5 mM, P < 0.05) but augmented by NCR (12.9 ± 2.0 mM, P < 0.05). In contrast, endocardial [K+]o, rise remained unaffected (7.6 ± 0.2 mM CTRL, 7.6 ± 1.3 mM GLIB, and 9.4 ± 2.2 mM NCR, P = NS). Conclusion: Activation of KATP channels plays an important role in epicardial [K+]o rise, but not in endocardial [K+]o rise, during regional ischemia. Another mechanism(s) may he important for endocardial [K+]o accumulation. 相似文献
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