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Focal Arrhythmia Ablation Determined by High‐Resolution Noninvasive Maps: Multicenter Feasibility Study
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MÉLÈZE HOCINI M.D. ASHOK J. SHAH M.D. THOMAS NEUMANN M.D. MALTE KUNISS M.D. DAMIR ERKAPIC M.D. ARNAUD CHAUMEIL M.D. SHAHNAZ‐JAMIL COPLEY M.D. PHANG BOON LIM M.D. PRAPA KANAGARATNAM M.D. ARNAUD DENIS M.D. NICOLAS DERVAL M.D. RÉMI DUBOIS M.D. HUBERT COCHET M.D. PIERRE JAIS M.D. MICHEL HAISSAGUERRE M.D. 《Journal of cardiovascular electrophysiology》2015,26(7):754-760
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THOMAS NEUMANN M.D. MALTE KUNISS M.D. GUIDO CONRADI M.D. JOHANNES SPERZEL M.D. ALEXANDER BERKOWITSCH Ph .D. SERGEY ZALTSBERG M.D. MACIEJ WOJCIK M.D. DAMIR ERKAPIC M.D. THORSTEN DILL M.D. CHRISTIAN W. HAMM M.D. F.E.S.C. HEINZ-F. PITSCHNER M.D. F.E.S.C. 《Journal of cardiovascular electrophysiology》2009,20(3):251-257
Introduction: Severe pulmonary vein stenosis (PVS) after catheter ablation of atrial fibrillation (AF) is a well-recognized complication with a further reported incidence of 1.3%. The preferred therapy for symptomatic PVS is pulmonary vein (PV) angioplasty, but this treatment modality is followed by restenosis in 44–70%. Whether there is additional long-term benefit from PVS stenting is uncertain. The aim of this study was the evaluation of the long-term success after PV stenting of severe stenosis.
Methods and Results: Ten patients (pts) with 13 PVS were prospectively evaluated. PV stenting was performed with Palmaz Genesis stents. Magnetic resonance imaging (MRI), lung perfusion scans, and CT-scans were performed before, directly after, and every 12 months thereafter. Primary endpoint of the study was the occurrence of restenosis after PV stenting. After a median follow-up of 47.7 (IQRs 25/75 47.2–48.5) months, the primary endpoint was achieved in 3 out of 13 PVs (23% of the treated PVs). We observed two in-stent restenosis 2 and 4 years after PV stenting. These pts experienced onset of dyspnea some weeks before. After an additional balloon angioplasty, the in-stent restenosis was resolved. In one asymptomatic patient, we observed an occlusion of the PV stent 13 months poststenting. Normalization of lung perfusion was noted 4 years after PV stenting versus directly poststenting in all pts without in-stent restenosis (n = 7).
Conclusion: PVS stenting with stent sizes ≥10 mm seems to be an adequate therapy modality for treatment of severe acquired PVS. Late in-stent restenosis after PVS stenting can occur. The normalization of the initially disturbed lung perfusion scan is possible and remains stable, even 4 years after PVS stenting. 相似文献
Methods and Results: Ten patients (pts) with 13 PVS were prospectively evaluated. PV stenting was performed with Palmaz Genesis stents. Magnetic resonance imaging (MRI), lung perfusion scans, and CT-scans were performed before, directly after, and every 12 months thereafter. Primary endpoint of the study was the occurrence of restenosis after PV stenting. After a median follow-up of 47.7 (IQRs 25/75 47.2–48.5) months, the primary endpoint was achieved in 3 out of 13 PVs (23% of the treated PVs). We observed two in-stent restenosis 2 and 4 years after PV stenting. These pts experienced onset of dyspnea some weeks before. After an additional balloon angioplasty, the in-stent restenosis was resolved. In one asymptomatic patient, we observed an occlusion of the PV stent 13 months poststenting. Normalization of lung perfusion was noted 4 years after PV stenting versus directly poststenting in all pts without in-stent restenosis (n = 7).
Conclusion: PVS stenting with stent sizes ≥10 mm seems to be an adequate therapy modality for treatment of severe acquired PVS. Late in-stent restenosis after PVS stenting can occur. The normalization of the initially disturbed lung perfusion scan is possible and remains stable, even 4 years after PVS stenting. 相似文献
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Safety of the Wearable Cardioverter Defibrillator (WCD) in Patients with Implanted Pacemakers
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SEBASTIEN JANIN M.D. MACIEJ WOJCIK M.D. MALTE KUNISS M.D. ALEXANDER BERKOWITSCH Ph.D. DAMIR ERKAPIC M.D. SERGEY ZALTSBERG M.D. FIONA ECARNOT
M.Sc. CHRISTIAN W. HAMM M.D. HEINZ F. PITSCHNER M.D. THOMAS NEUMANN M.D. 《Pacing and clinical electrophysiology : PACE》2010,33(7):784-789
Background: Delayed interatrial conduction, manifested on the electrocardiogram as a P wave ≥110 ms (interatrial block, IAB), is highly prevalent and associated with atrial fibrillation (AF). It is correlated with P‐terminal force (Ptf; product of the duration and amplitude of the negative terminal phase of the P wave in lead V1). Our purpose was to describe the modifications of the P‐wave duration and Ptf after pulmonary vein antrum isolation (PVAI) in patients with paroxysmal AF. Methods: PVAI was performed in 45 patients with paroxysmal AF, either with the cryoballoon technique (n = 15) or radiofrequency ablation (n = 30). Electrocardiograms were recorded before PVAI, 3 and 6 months after ablation. Results: From the sample (median age 60 [53; 66] years; female 40%), median P‐wave duration was 122 [114; 134] ms before PVAI and 116 [106; 124] ms at 3‐month follow‐up (P < 0.001). IAB was observed in 42 patients (93.3%) before ablation and in 31 patients (68.9%) at 3‐month follow‐up. Median Ptf was 0.047 [0.020; 0.068] before ablation and 0.013 [0.004; 0.025] at 3‐month follow‐up (P < 0.001). Twenty‐six patients (57.8%) had a Ptf > 0.04 mV x ms before ablation and only one (2.2%) at 3‐month follow‐up. P‐wave duration and Ptf were not significantly modified between 3‐ and 6‐month follow‐up. Conclusion: The terminal part of the P wave is modified after PVAI, perhaps due to the loss of pulmonary vein antrum signals. P‐wave duration and Ptf must be carefully interpreted after such a procedure. The prognostic value of these modifications should be evaluated. (PACE 2010; 784–789) 相似文献
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Applicability of a Novel Formula (Bogossian formula) for Evaluation of the QT‐Interval in Heart Failure and Left Bundle Branch Block Due to Right Ventricular Pacing
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GERRIT FROMMEYER HARILAOS BOGOSSIAN ELENI PECHLIVANIDOU PHILIPP CONZEN CHRISTOPHER GEMEIN KAY WEIPERT INGA HELMIG RITVAN CHASAN VICTORIA JOHNSON LARS ECKARDT CHRISTIAN W HAMM MELCHIOR SEYFARTH BERND LEMKE MARKUS ZARSE JÖRN SCHMITT DAMIR ERKAPIC 《Pacing and clinical electrophysiology : PACE》2017,40(4):409-416
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