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对90例心脏病患者进行心内膜心肌活检(EMB)95例次,成功率96.8%,临床诊断原因不明心律失常44例,各型心肌病24例,疑诊心肌炎10例,先心并心律失常4例,缩窄性心包炎5例,二尖瓣脱垂3例,经光、电镜检查确定诊断72例(80%)。其中心肌炎44例,扩张型心肌病19例、肥厚型1例,限制型4例,炎性心肌病4例。排除诊断3例(3.4%),无助于诊断15例(16.6%)。全组均无严重并发症,作者认为EMB对心肌疾病(尤其对心内膜心肌纤维化病)及心肌炎有较大诊断价值的新技术,值得推广。 相似文献
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目的探讨药物多巴酚丁胺、硝酸异山梨酯负荷试验核素锝[^99mTc]甲氧异丁异腈(technetium [^99mTc] methoxy isobutyl isonitrile,^99mTc-MIBI)心肌断层显像识别存活心肌的价值。方法对48例陈旧性心肌梗死伴左心室收缩功能受损患者进行静息心肌^99mTc-MIBI心肌断层显像和多巴酚丁胺、硝酸异山梨酯负荷试验^99mTc-MIBI心肌断层显像,用半定量的方法将^99mTc-MIBI摄取进行评分,区分存活心肌和非存活心肌。结果^99mTc-MIBI显像判定,经皮冠状动脉介入术前存活心肌节段有279个.非存活心肌节段235个;经多巴酚丁胺加硝酸异山梨酯负荷后,心肌显像改善:轻度稀疏节段9个、中度稀疏节段24个、严重稀疏或缺损节段10个,共计43个节段显像改善,评分减少1分以上,存活心肌节段313个,非存活心肌节段201个。两组差异有统计学意义(P〈0.05)。经皮冠状动脉介入术后6个月,随访多巴酚丁胺加硝酸异山梨酯负荷后,心肌显像改善的32个节段发现,静息^99mTc-MIBI显像改善,另有8个受损节段正常化。多巴酚丁胺加硝酸异山梨酯负荷后心肌显像无变化的389个节段在经皮冠状动脉介入术后346个节段无变化。多巴酚丁胺加硝酸异山梨酯负荷心肌显像检测存活心肌的阳性预测值93%,阴性预测值89%。结论多巴酚丁胺加硝酸异山梨酯负荷后心肌显像对存活心肌的识别能够提高存活心肌的检出率。其阳性预测值和阴性预测值较好。 相似文献
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二尖瓣狭窄合并左心房血栓患者的球囊扩张术探讨 总被引:1,自引:0,他引:1
目的:探讨二尖瓣狭窄合并左心房血栓患者安全接受二尖瓣球囊扩张术(PBMV)的方法。方法:对二尖瓣狭窄合并左心房血栓的29例患者,给予正规的华法林抗凝治疗3~12个月,然后再施行PBMV。结果:华法林抗凝治疗后有24例患者的左心房血栓消失,23例患者的血栓消失的时间是6个月以内:5例患者的血栓均有不同程度的缩小并呈现高强度回声。对血栓消失的24例患者及缩小机化的5例患者成功地进行了PBMV;所有患者 相似文献
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The panax notoginseng saponin(PNS) had been clinically used for the treatment of cardiovascular diseases and stroke in China.It had been demonstrated that PNS could protect cardiomyocytes from injury induced by ischemi- a,but the underlying molecular mechanisms of this protective effect were still unclear.This study was aimed to investigate the protective effect and molecular mechanisms of PNS on apoptosis in H9c2 cells in vitro and rat myocardial ischemia injury model in vivo.Annexin-V/PI assay shew that PNS could protect H9c2 cells from apoptosis induced by serum, glucose and oxygen deprivation(SGOD) in a dose-dependent manner.However,the anti-apoptotic effect of PNS was reversed by LY294002,a specific PI3K inhibitor.This antiapoptotic effect of PNS was confirmed by JC-1,a specific probe of mitochondrial membrane potential staining.PNS could significantly increase phos-Akt in H9c2 cells by Western blot assays and its effect could be inhibited by LY294002.Furthermore,PNS could improve ischemic-induced left ventricular function as reflected by EF,LVDd and LVDs.PNS could also inhibited cellular apoptosis in myocardial tissues in ischemic rats by TUNEL assay.PNS administration also increased the expression of phos-Akt in rat ischemic myocardial tissues.These results suggested that PNS could protect myocardial cells from apoptosis induced by ischemia in vitro model and in vivo model through activating-PI3K/Akt signal pathway which may be meaningful for further understanding the molecular mechanisms of cardiac protection of PNS.And the results might be useful in treatment of myocardial ischemia in future. 相似文献
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为探讨影响心钠素(ANP)和内源性类洋地黄物质分泌的因素,我们研究了26例患者二尖瓣球囊扩张术(PBMC)前后血浆ANP和EDLS与心率(HR)、心输出量(CO)、心脏指数(CI)、平均右房压(MRAP)、平均左房压(MLAP)、平均二尖瓣跨瓣压差(MPG)和二尖瓣口面积(MVA)的变化和关系。结果显示,术后血浆ANP水平从316.92±103.41(与对照组113.90±33.52 pg/ml比较,P<0.001)降到205.96±64.50 pg/ml(与术前及对照组比较,P<0.001)。血浆EDLS水平也从323.04±231.25(与对照组113.38±61.47 pg/ml比较,P<0.001)降到145.92±96.97pg/ml(与术前比较P<0.001,与对照组比较P>0.1)。血浆ANP水平与HR、MRAP、MLAP和MPG呈显著正相关(r=0.784、0.389、0.819和0.435,P<0.001、0.005、0.001和0.002),与CO和MVA呈显著负相关(r=-0.294和-0.366,P<0.05和0.01)。血浆EDLS水平与HR、MLAP和MPG呈显著正相关(r=0.761、0.356和0.342,P<0.001、0.01和0.02)。血浆ANP与EDLS水平无显著相关(r=0.259,P>0.05)。 相似文献
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风湿性心脏病二尖瓣狭窄患者随着病情的进展常并发心房颤动(房颤),房颤使左心房血栓的发生率增高。左心房血栓常导致严重的周围动脉栓塞并发症,同时也限制了经皮二尖瓣球囊成形术(PBMV)[1]。本文对风心病二尖瓣狭窄合并左心房新鲜血栓的患者,给予正规的华法令抗凝治疗,然后再施行PBMV,收到满意效果,避免了开胸手术,现报道如下。1 临床资料和方法1.1 病例资料 经食管超声心动图(TEE)证实为风心病二尖瓣狭窄合并左心房血栓的患者41例。男10例,女31例。年龄18~65岁,平均38岁。40例为房颤,房颤病史1~15年;1例为窦性心律。心功能… 相似文献