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排序方式: 共有208条查询结果,搜索用时 15 毫秒
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Binu Prathap Thomas Sreekanth Raveendran Thenmozhi Mani 《Indian Journal of Orthopaedics》2021,55(2):478
BackgroundTardy ulnar nerve palsy is the development of late onset ulnar nerve dysfunction and is usually treated by open anterior transposition of ulnar nerve. Open technique is done using a longitudinal incision about 6–8 inch. in length with chances of development of medial antebrachial cutaneous nerve neuromas.PurposeIn this study, we describe the technique of Endoscopic Anterior Transposition of Ulnar Nerve (EATUN procedure) to treat tardy ulnar nerve palsy and analyze the results.MethodsSeven patients diagnosed to have tardy ulnar nerve palsy was treated by EATUN. The humerus-elbow-wrist angle (HEW), pre- and post-operative intrinsic muscle power and sensory assessment, Dellon scores, and the Q-DASH was analyzed.ResultsThe minimum follow-up was 12 months (Mean 27.4 months, Range 12–36 months). Improvement in Dellon and Q-DASH scores following EATUN procedure was statistically significant. There was objective improvement of intrinsic muscle power and sensation on follow-up, though not statistically significant. No instance of neuroma of the medial cutaneous nerve of forearm was noted.ConclusionsThe endoscopic anterior transposition of the ulnar nerve is a good option in surgical management of tardy ulnar nerve palsy.Level of evidenceTherapeutic Level IV.Supplementary InformationThe online version contains supplementary material available at 10.1007/s43465-021-00366-w. 相似文献
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Sreeja L Syamala VS Syamala V Hariharan S Raveendran PB Vijayalekshmi RV Madhavan J Ankathil R 《Journal of cancer research and clinical oncology》2008,134(6):645-652
Purpose Inter individual variation in lung cancer susceptibility may be modulated in part through genetic polymorphisms in the DNA
repair genes, especially the genes involved in the Base Excision Repair (BER) and nucleotide excision repair (NER) pathway.
Two of the genetic polymorphisms, XRCC1Arg399Gln and XPD Lys751Gln have been extensively studied in the association with lung
cancer risk, although published studies have been inconclusive.
Methods In order to verify the role of the common variant alleles in the XPD gene, we have genotyped 211 lung cancer patients and
211 healthy controls using PCR-RFLP assays in a hospital based, case-control study in an Indian population. Logistic regression
models were fit to examine the relationship between the log odds of lung cancer and each covariate. Overall Survival in relation
to various genotypes and clinicopathological factors were analyzed using Kaplan Meier estimates and hazard ratios were calculated
using Cox Regression analysis.
Results The carriers of XRCC1 399 AA genotypes were at higher risk of lung cancer (OR = 2.1, 95% CI:1.224–3.669, P = 0.007) than carriers
of GG genotype. Subjects carrying 751 AC genotype were at an increased risk of carcinoma of the lung (OR = 1.8; 95% CI:1.233–2.807,
P = 0.003) than subjects with AA genotypes. Compared to the XRCC1 399 GG/ XPD 751 AA reference genotype, the combined variants,
XRCC1 399 GG/ XPD 751 AC+CC (OR = 1.9, 95% CI: 1.037–3.481), P = 0.03), XRCC1 399 GA+AA/ XPD 751 AA (OR = 1.7, 95% CI: 1.020–2.833,
P = 0.04), XRCC1 399 GA+AA/XPD 751 AC+CC (OR = 2.7, 95% CI: 1.582–4.864, P = 0.01), had significantly higher odds ratios.
Increasing numbers of either XPD or XRCC1 variant alleles were associated with shorter overall survival, the risk being significant
for the XRCC1 gene polymorphism (P = 0.01 by log-rank test). The hazard of dying was significant for the XRCC1 399 AA genotype
(HR = 3.04, 95%CI: 1.393–6.670, P = 0.005). Higher tumour stage also came out as significant predictors of patient death.
Conclusions These findings suggest that genetic polymorphisms in the DNA repair genes may modulate overall lung cancer susceptibility
and that pathological stage and XRCC1 Arg399Gln independently predicted overall survival among Indian lung cancer patients. 相似文献
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Lauren L. O'Mahoney PhD Patrick J. Highton PhD Laura Kudlek MSc Jessica Morgan BA Rosie Lynch BA Ella Schofield BMBCh Nayanika Sreejith BA Ajay Kapur BMBCh Afolarin Otunla BA Sven Kerneis BMBCh Olivia James BA Karen Rees PhD Ffion Curtis PhD Kamlesh Khunti FMedSci Jamie Hartmann-Boyce DPhil 《Diabetes, obesity & metabolism》2022,24(9):1850-1860
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K. Raveendran Pillai K. Sujathan S. Kannan Elizabeth K. Abraham Babu Mathew N. Sreedevi Amma M. Krishnan Nair Venugopal P. Menon 《Journal of cancer research and clinical oncology》1994,120(12):723-726
The present study has analysed the numbers of argyrophilic nucleolar organizer regions (AgNOR) in normal tissues and in premalignant and malignant lesions of the oral mucosa in order to assess their potential as a biological marker for tumour progression. On comparison of AgNOR numbers in different lesions, carcinomas showed the highest number (4.65±0.98) compared to leukoplakias (2.38±0.47) and normal tissues (1.53±0.39). Spindle cell carcinomas and poorly differentiated squamous cell carcinomas had higher AgNOR counts than well-differentiated carcinomas. In various clinically different types of oral leukoplakia, the lowest AgNOR counts were observed in homogenous leukoplakia and the highest in speckled leukoplakia. No significant difference in AgNOR number was observed between non-dysplastic and dysplastic leukoplakia, although a significant difference was evident between dysplastic leukoplakia and normal oral mucosa. Correlating the AgNOR count and tumour progression, a significantly high positive correlation coefficient (r=0.7969,P=0.0000) was observed. 相似文献
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Deepa Joseph Dubashi Biswajit Rajesh Nachiappa Ganesh Sreejith Parameswaran Ankit Jain 《Indian journal of hematology & blood transfusion》2014,30(1):45-47
Renal involvement is frequent in hematologic malignancies especially Hodgkin’s lymphoma. Renal complications in children with malignancies primarily arise from tumour lysis syndrome, malignant infiltration or obstruction of the urinary tract, deposits of immunoglobulin fractions or crystals, renal infiltration by malignant cells, paraneoplastic or storage glomerulopathies. Nephrotic syndrome has been described in B cell type Non Hodgkin’s lymphomas. There are very few reports of association of T cell lymphoma and nephrotic syndrome in pediatric patients. We present a case of peripheral T cell lymphoma with nephrotic syndrome in a 10 year old boy. 相似文献
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