采用电针鉴别分型治疗周围性面神经麻痹

通过患侧太阳、阳白穴对电针的感应程度进行分型，在明确分型的基础上对周围性面神经麻痹患358例进行针对性治疗。治愈率一般型为93．4％，顽固型为28．3％。总有效率分别为100％和96．2％。     

TENS镇痛及影响因素

TENS镇痛是将表面电极放置在痛区皮肤或与痛区相关穴位的表面,用连续电脉冲刺激外周感受器,造成组织对伤害性刺激的感受性降低,达到镇痛的目的.TENS镇痛的机理可能是通过阈下刺激引起伤害感受性传入纤维的电紧张性极化作用,导致传导阻滞;TENS 的强度往往只兴奋A类纤维通过闸门控制,阻断伤害性信息向中枢传导;通过激活痛抑制系统、抑制三叉神经脊束核Ⅰ、Ⅱ层中由伤害性刺激诱导的SP释放的增加;患者心理因素的作用.电极位置、电脉冲模式、个人对痛觉的感知和对疼痛强度的评价与个人当时的心理状态和以往的体验等影响TENS 的镇痛效果.     

52例针灸结合刺络放血治疗骨性膝关节炎临床观察及护理

[目的]观察针灸结合刺络放血法治疗骨性膝关节炎的临床疗效和护理效果.[方法]对52例骨性膝关节炎病人采用针灸结合刺络放血治疗,并给予活动与功能锻炼指导,将治疗前和治疗20 d后病人疼痛程度、晨僵、肿胀、日常步行活动能力以及疼痛缓解时间进行分级评分比较.[结果]治疗前后各项指标的差异均有统计学意义(P＜0.01).[结论]针灸配合刺络放血法结合活动指导及功能锻炼治疗骨性膝关节炎,能够快速缓解病人关节疼痛、肿胀、晨僵,改善步行能力,从而提高生活质量.     

颈丛臂丛联合神经阻滞在锁骨切开复位内固定术中的应用

目前锁骨骨折行切开复位内固定术的病例呈增加趋势。而对该种手术采用的麻醉方法较多。近 2年来 ,作者对锁骨骨折行切开复位内固定术采用颈丛臂丛联合神经阻滞麻醉 ,并与单纯颈丛神经阻滞麻醉和单纯臂丛神经阻滞麻醉方法进行了比较 ,结果报道如下。1　资料和方法1.1　对象与分组　随机选择锁骨骨折须行切开复位内固定术的病人 6 0例 ,年龄 2 1～ 6 3岁 ,体重 4 0～10 0kg ,手术前无严重的呼吸、循环系统疾患或严重肝肾疾病 ,ASAⅠ～Ⅱ级 ,采用随机双盲法 ,将病人分成三组 ,每组 2 0例。Ⅰ组为单纯颈丛神经阻滞组 ;Ⅱ组为单纯臂丛神经阻滞…     

腰神经后支电针治疗腰腿痛

最近读到于云、程关群撰写的腰神经后支阻滞治疗腰腿痛83例报告,介绍的阻滞治疗方法有效率为89．15％,患者易于接受,能迅速起效止痛、恢复正常的生活和工作,疗效是肯定的,值得借鉴。日前笔者会诊1例车祸导致腰腿痛的患者,参照报告中的腰神经后支进针位置图,试用电针治疗。该例,男,58岁,农民,横穿马路时不慎被汽车撞倒,头部裂伤出血,腰腿疼痛不能活动。急送本院清创缝合     

大椎穴合谷刺配合夹脊治疗颈椎病

颈椎病又称颈椎综合征，是由于退变、增生的间盘、骨赘压迫临近的血管、神经引起的病变，由于压迫的部位不同，临床上分为神经根型、椎动脉形、脊髓型、交感神经型、混合型（至少包括以上两种类型）。神经根型表现：颈部僵硬、活动受限、上肢麻木、疼痛、浅感觉迟钝、肌力减退、反射弱；椎动脉型表现：头痛、头晕脑血管痉挛症状及眩晕、恶心、耳鸥、视物不清等脑缺血症状。本人近年来采用大椎穴合谷剌配合夹脊穴治疗椎动脉型、神经根型及两重混合型的颈椎病48例，取得满意疗效，现介绍如下。     

以电针为主综合治疗单纯性肥胖55例临床观察

当人体进食热量多于消耗能量时,多余热量就以脂肪形式储存于体内,其量超过正常生理需求,且达到一定值时遂成为肥胖症(obesity)。     

浅刺为主分期治疗周围性面瘫39例

周围性面瘫为一种临床常见的疾病,西医认为本病多由急性非化脓性面神经炎以及面部受风吹或着凉所引起,可能是局部营养神经的血管痉挛,导致该神经缺血、水肿,亦可能与局部     

电针治疗腰三横突综合征72例临床体会

目的:观察针灸加电刺激治疗腰三横突综合征患者的临床疗效。方法:对72例腰三横突综合征的患者进行电针疗法的观察治疗。结果:总有效率96.7%。结论:通过电针,可健腰补肾、活血化瘀共奏止腰痛之效。     

Effect of electro-acupuncture combined with early rehabilitation on motor function and expressions of CD11b/CD18 and tumor necrosis factor-α in patients with acute cerebral infarction

Objective To observe the effect of electro-acupuncture combined with early rehabilitation on the motor function and expressions of the adhesion molecules CD11b and CD18 in the polymorphonuclear leucocytes (PMN) and monocytes and serum tumor necrosis factor-α (TNF-α) levels in patients with acute cerebral infarction (ACI). Methods A total of 165 ACI patients were randomly divided into control group (group A, n=50), conventional rehabilitation group (group B, n=50) and comprehensive rehabilitation group (group C, n=65). The expressions of CD11b and CD18 in the PMN and monocytes and serum TNF-α levels were determined before and at 1, 2, and 4 weeks after the treatment. Thirty-two healthy subjects were also recruited as the normal control group (group N). The neurological function of the subjects was evaluated by modified Edinburgh-Scandinavia stroke scale (MESSS) and Fugi-Meyer Assessment (FMA), and their activity of daily living (ADL) was assessed using Barthel index (BI). Results The CD11b/CD18 expression in the PMN and MN and serum TNF-α level in groups A, B and C were significantly higher than those in group N before and 1 week after the treatment (P<0.05). CD11b/CD18 expression and serum TNF-α level were significantly lower in groups B and C than in the group A at 1 week after the treatment, and significantly lower in group C than in group B (P<0.05). At 2 weeks of treatment, CD11b/CD18 and TNF-α were significantly lower in groups B and C than in the group A, being the lowest in group C (P<0.05). The scores of mESSS in both groups B and C were lower than that in group A, and the scores were lower in group C than in group B. Group C showed higher FMA scores than group B, both having higher scores than group A. At 4 weeks of treatment, the mESSS scores were significantly lower, hut the FMA and ADL score significantly higher in groups B and C than in the control group (P<0.05), and the differences were more obvious in group C. Groups B and C had greater effective rate than group A (P<0.05), and the rate was the highest in group C (P<0.05). Conclusion Electro-acupuncture combined with early rehabilitation promotes the recovery of motor function in ACI patients probably by regulating the expressions of the adhesion molecules CD11b and CD18 on the PMN and monocytes and the serum levels of TNF-α.