脊髓P物质在电针镇痛中作用的研究进展

脊髓P物质是伤害性传入末梢释放的一种兴奋性递质,它既可传递痛觉信息,又有镇痛作用。在脊髓水平参与痛觉调控,而电针在脊髓水平可能是通过内源性阿片肽抑制SP而产生镇痛作用。本文对近十年来脊髓P物质参与痛觉调控及在电针镇痛中的作用的研究状况作了系统综述。     

利用小样本预处理技术提高敏感性问题调查精度

本文首先尝试用小样本预处理技术——熵判别方法处理和判别敏感性问题调查中不同调查方法的调查结果中的粗大误差,然后用捕获-再捕获方法(CRM)校正调查结果的数值误差,提高敏感性问题抽样调查结果的可靠性。原理与方法(一)熵判别法熵是信息论中的一个基本概念,在信息论中信息量I(Ak)表示观测到一个以概率pk发生的事件Ak的信息。定义I(xk)=ln(p1k)=-lnpk,定义熵为H(xk)=E{I(xk)}=E[-lnpk],熵与方差之间存在一定的对应关系H(x)=ln(ασ)其中α为与分布有关的常数,σ为标准差。由上式可以看出,x取值的分散程度越大,其熵越大,因为熵是对不…     

推拿、穴位注射配合电针治疗颈椎病

近几年来我们采用推拿、穴位注射配合电针治疗颈椎病收到了较好的效果,现报道如下. 一般资料 本组病例共60例,其中门诊40例,住院20例;     

采用电针鉴别分型治疗周围性面神经麻痹

通过患侧太阳、阳白穴对电针的感应程度进行分型，在明确分型的基础上对周围性面神经麻痹患358例进行针对性治疗。治愈率一般型为93．4％，顽固型为28．3％。总有效率分别为100％和96．2％。     

TENS镇痛及影响因素

TENS镇痛是将表面电极放置在痛区皮肤或与痛区相关穴位的表面,用连续电脉冲刺激外周感受器,造成组织对伤害性刺激的感受性降低,达到镇痛的目的.TENS镇痛的机理可能是通过阈下刺激引起伤害感受性传入纤维的电紧张性极化作用,导致传导阻滞;TENS 的强度往往只兴奋A类纤维通过闸门控制,阻断伤害性信息向中枢传导;通过激活痛抑制系统、抑制三叉神经脊束核Ⅰ、Ⅱ层中由伤害性刺激诱导的SP释放的增加;患者心理因素的作用.电极位置、电脉冲模式、个人对痛觉的感知和对疼痛强度的评价与个人当时的心理状态和以往的体验等影响TENS 的镇痛效果.     

腰神经后支电针治疗腰腿痛

最近读到于云、程关群撰写的腰神经后支阻滞治疗腰腿痛83例报告,介绍的阻滞治疗方法有效率为89．15％,患者易于接受,能迅速起效止痛、恢复正常的生活和工作,疗效是肯定的,值得借鉴。日前笔者会诊1例车祸导致腰腿痛的患者,参照报告中的腰神经后支进针位置图,试用电针治疗。该例,男,58岁,农民,横穿马路时不慎被汽车撞倒,头部裂伤出血,腰腿疼痛不能活动。急送本院清创缝合     

Effects of hypoxic preconditioning on the changes of expression of neuroglobin mRNA and labeled positive cells following cerebral ischemia in gerbils

INTRODUCTION Neuroglobin (NGB) is a recently discovered member of the vertebrate globin family [1], predominantly expressed in various brain regions of human and vertebrate. The expression of NGB was increased in case of ischemia, which may play a role in…     

以电针为主综合治疗单纯性肥胖55例临床观察

当人体进食热量多于消耗能量时,多余热量就以脂肪形式储存于体内,其量超过正常生理需求,且达到一定值时遂成为肥胖症(obesity)。     

电针治疗腰三横突综合征72例临床体会

目的:观察针灸加电刺激治疗腰三横突综合征患者的临床疗效。方法:对72例腰三横突综合征的患者进行电针疗法的观察治疗。结果:总有效率96.7%。结论:通过电针,可健腰补肾、活血化瘀共奏止腰痛之效。     

Effect of electro-acupuncture combined with early rehabilitation on motor function and expressions of CD11b/CD18 and tumor necrosis factor-α in patients with acute cerebral infarction

Objective To observe the effect of electro-acupuncture combined with early rehabilitation on the motor function and expressions of the adhesion molecules CD11b and CD18 in the polymorphonuclear leucocytes (PMN) and monocytes and serum tumor necrosis factor-α (TNF-α) levels in patients with acute cerebral infarction (ACI). Methods A total of 165 ACI patients were randomly divided into control group (group A, n=50), conventional rehabilitation group (group B, n=50) and comprehensive rehabilitation group (group C, n=65). The expressions of CD11b and CD18 in the PMN and monocytes and serum TNF-α levels were determined before and at 1, 2, and 4 weeks after the treatment. Thirty-two healthy subjects were also recruited as the normal control group (group N). The neurological function of the subjects was evaluated by modified Edinburgh-Scandinavia stroke scale (MESSS) and Fugi-Meyer Assessment (FMA), and their activity of daily living (ADL) was assessed using Barthel index (BI). Results The CD11b/CD18 expression in the PMN and MN and serum TNF-α level in groups A, B and C were significantly higher than those in group N before and 1 week after the treatment (P<0.05). CD11b/CD18 expression and serum TNF-α level were significantly lower in groups B and C than in the group A at 1 week after the treatment, and significantly lower in group C than in group B (P<0.05). At 2 weeks of treatment, CD11b/CD18 and TNF-α were significantly lower in groups B and C than in the group A, being the lowest in group C (P<0.05). The scores of mESSS in both groups B and C were lower than that in group A, and the scores were lower in group C than in group B. Group C showed higher FMA scores than group B, both having higher scores than group A. At 4 weeks of treatment, the mESSS scores were significantly lower, hut the FMA and ADL score significantly higher in groups B and C than in the control group (P<0.05), and the differences were more obvious in group C. Groups B and C had greater effective rate than group A (P<0.05), and the rate was the highest in group C (P<0.05). Conclusion Electro-acupuncture combined with early rehabilitation promotes the recovery of motor function in ACI patients probably by regulating the expressions of the adhesion molecules CD11b and CD18 on the PMN and monocytes and the serum levels of TNF-α.