肝硬化合并上消化道出血108例的护理

目的:探讨肝硬化合并上消化道出血的临床护理和预见性观察病情方法.方法:回顾分析108例肝硬化合并上消化道出血的护理体会.结果:本组痊愈出院96例,自动出院7例,死亡5例.死亡原因:失血性休克3例,肝性脑病引起肝肾综合征1例,肝性脑病合并感染1例.结论:加强临床急救和预见性护理可以增加治愈率,减少上消化道出血后肝性脑病和继发性感染的发生;有效的心理、饮食护理和健康教育可以减少并发症的反复发作.     

地塞米松预防剖宫产患者自控镇痛不良反应的临床研究

目的观察地塞米松预防剖宫产患者自控镇痛致恶心、呕吐、瘙痒等不良反应疗效。方法选择同期妇产科硬膜外麻醉手术患者。I组（实验组）：剖宫产手术50例，Ⅱ组（对照组）：妇科手术50例。两组于手术结束时施行患者自控硬膜外镇痛，同时I组静脉注射地塞米松10mg；Ⅱ组静脉注射格拉司琼4mg。观察记录患者镇痛效果（视觉模拟评分法）及恶心、呕吐、胃肠恢复、瘙痒等不良反应的发生率。结果镇痛效果确切，两组比较差异无统计学意义（P〉0.05），不良反应：恶心、呕吐发生率两组差异无统计学意义（P〉0.05），皮肤瘙痒发生率及胃肠恢复延长I组低于Ⅱ组（P〈0.05）。结论地塞米松能有效预防剖宫产患者自控镇痛所致恶心、呕吐、皮肤瘙痒等不良反应。     

阿替洛尔治疗急性心肌梗塞90例临床分析

急性心肌梗塞(AMI)是一种危急状态,病死率高。我院自1994年来,采用选择性β1-受体阻滞剂阿替洛尔治疗AMI90例,效果良好,现予报道。     

克山病患者抗氧化能力测定

Objective To explore the relationship between myocardial damage and antioxidant capacity in vivo of Keshan disease patients and to analyze the possible pathogenesis. Methods In the period from 2005 to 2006, 41 chronic and latent Keshan disease cases were chosen as the case group from such serious endemic areas as Yongjin Village, Xinfa Village of Fuyu County, Fuan Village of Shangzhi County, Xinghuo Village of Wudalianchi County, 61 healthy people from the same area as internal controls, 48 healthy people from Xianglansan Village of Wangkui County, an un-endemic area, as external control. Fasting peripheral venous blood was collected from all the people. And blood selenium, glutathione peroxidase(GSH-Px) and suporoxide dismutase.(T-SOD) activities, malondialdehyde (MDA) levels were examined. Results Blood selenium level of the patient group [(34.80±13.30) μg/L], GSH-Px[(104.10±34.19)U/L]and T-SOD[(92.16±17.98)×103 U/L]actives were significantly lower than the internal control group [(41.24±13.57)μg/L, (118.57±25.49)U/L, (104.82±13.56)×103 U/L]and the external control group [(48.33±16.51)μg/L, (155.00±24.01)U/L, (108.48±12.73)×103 U/L], respectively, with a statistically significant difference(all P < 0.05). MDA level of the patient group[(7.12± 1.37)μmol/L]was higher than that in the internal control group[(5.36±1.18)μmol/L]and the external control group[(5.22±0.83)μmol/L]with a statistically significant differences(both P < 0.05). The blood selenium level, GSH-Px activity of internal control group was resoectively lower than that in the external control group, the differences being statistically significant(beth P < 0.05). Conclusions Selenium deficiency may lead to reduced antioxidant capacity and enhanced oxidative damage in Keshan disease patients in vivo. There may be a certain relationship between oxidative stress disorder and myocardial damage of Keshan disease.     

独针后溪穴治疗颈肩腰腿痛临床体会

笔者在针刺后溪穴的同时让患者带针在生理活动范围内尽力活动，以治疗颈肩腰腿痛获得满意效果，介绍如下。 1适用范围 颈痛如失枕、颈椎病、颈椎间盘突出症，肩痛如肩周炎、颈椎性肩痛症，手痛如网球肘、腕管综合征，腰痛如急性腰扭伤、慢性腰肌劳损、腰椎病、腰三横突综合征、腰椎间盘突出症，腿痛如坐骨神经痛、梨状肌综合征、退行性膝关节炎、急性踝扭伤、跟痛症。     

克山病患者抗氧化能力测定

Objective To explore the relationship between myocardial damage and antioxidant capacity in vivo of Keshan disease patients and to analyze the possible pathogenesis. Methods In the period from 2005 to 2006, 41 chronic and latent Keshan disease cases were chosen as the case group from such serious endemic areas as Yongjin Village, Xinfa Village of Fuyu County, Fuan Village of Shangzhi County, Xinghuo Village of Wudalianchi County, 61 healthy people from the same area as internal controls, 48 healthy people from Xianglansan Village of Wangkui County, an un-endemic area, as external control. Fasting peripheral venous blood was collected from all the people. And blood selenium, glutathione peroxidase(GSH-Px) and suporoxide dismutase.(T-SOD) activities, malondialdehyde (MDA) levels were examined. Results Blood selenium level of the patient group [(34.80±13.30) μg/L], GSH-Px[(104.10±34.19)U/L]and T-SOD[(92.16±17.98)×103 U/L]actives were significantly lower than the internal control group [(41.24±13.57)μg/L, (118.57±25.49)U/L, (104.82±13.56)×103 U/L]and the external control group [(48.33±16.51)μg/L, (155.00±24.01)U/L, (108.48±12.73)×103 U/L], respectively, with a statistically significant difference(all P < 0.05). MDA level of the patient group[(7.12± 1.37)μmol/L]was higher than that in the internal control group[(5.36±1.18)μmol/L]and the external control group[(5.22±0.83)μmol/L]with a statistically significant differences(both P < 0.05). The blood selenium level, GSH-Px activity of internal control group was resoectively lower than that in the external control group, the differences being statistically significant(beth P < 0.05). Conclusions Selenium deficiency may lead to reduced antioxidant capacity and enhanced oxidative damage in Keshan disease patients in vivo. There may be a certain relationship between oxidative stress disorder and myocardial damage of Keshan disease.     

克山病患者抗氧化能力测定

Objective To explore the relationship between myocardial damage and antioxidant capacity in vivo of Keshan disease patients and to analyze the possible pathogenesis. Methods In the period from 2005 to 2006, 41 chronic and latent Keshan disease cases were chosen as the case group from such serious endemic areas as Yongjin Village, Xinfa Village of Fuyu County, Fuan Village of Shangzhi County, Xinghuo Village of Wudalianchi County, 61 healthy people from the same area as internal controls, 48 healthy people from Xianglansan Village of Wangkui County, an un-endemic area, as external control. Fasting peripheral venous blood was collected from all the people. And blood selenium, glutathione peroxidase(GSH-Px) and suporoxide dismutase.(T-SOD) activities, malondialdehyde (MDA) levels were examined. Results Blood selenium level of the patient group [(34.80±13.30) μg/L], GSH-Px[(104.10±34.19)U/L]and T-SOD[(92.16±17.98)×103 U/L]actives were significantly lower than the internal control group [(41.24±13.57)μg/L, (118.57±25.49)U/L, (104.82±13.56)×103 U/L]and the external control group [(48.33±16.51)μg/L, (155.00±24.01)U/L, (108.48±12.73)×103 U/L], respectively, with a statistically significant difference(all P < 0.05). MDA level of the patient group[(7.12± 1.37)μmol/L]was higher than that in the internal control group[(5.36±1.18)μmol/L]and the external control group[(5.22±0.83)μmol/L]with a statistically significant differences(both P < 0.05). The blood selenium level, GSH-Px activity of internal control group was resoectively lower than that in the external control group, the differences being statistically significant(beth P < 0.05). Conclusions Selenium deficiency may lead to reduced antioxidant capacity and enhanced oxidative damage in Keshan disease patients in vivo. There may be a certain relationship between oxidative stress disorder and myocardial damage of Keshan disease.     

克山病患者抗氧化能力测定

Objective To explore the relationship between myocardial damage and antioxidant capacity in vivo of Keshan disease patients and to analyze the possible pathogenesis. Methods In the period from 2005 to 2006, 41 chronic and latent Keshan disease cases were chosen as the case group from such serious endemic areas as Yongjin Village, Xinfa Village of Fuyu County, Fuan Village of Shangzhi County, Xinghuo Village of Wudalianchi County, 61 healthy people from the same area as internal controls, 48 healthy people from Xianglansan Village of Wangkui County, an un-endemic area, as external control. Fasting peripheral venous blood was collected from all the people. And blood selenium, glutathione peroxidase(GSH-Px) and suporoxide dismutase.(T-SOD) activities, malondialdehyde (MDA) levels were examined. Results Blood selenium level of the patient group [(34.80±13.30) μg/L], GSH-Px[(104.10±34.19)U/L]and T-SOD[(92.16±17.98)×103 U/L]actives were significantly lower than the internal control group [(41.24±13.57)μg/L, (118.57±25.49)U/L, (104.82±13.56)×103 U/L]and the external control group [(48.33±16.51)μg/L, (155.00±24.01)U/L, (108.48±12.73)×103 U/L], respectively, with a statistically significant difference(all P < 0.05). MDA level of the patient group[(7.12± 1.37)μmol/L]was higher than that in the internal control group[(5.36±1.18)μmol/L]and the external control group[(5.22±0.83)μmol/L]with a statistically significant differences(both P < 0.05). The blood selenium level, GSH-Px activity of internal control group was resoectively lower than that in the external control group, the differences being statistically significant(beth P < 0.05). Conclusions Selenium deficiency may lead to reduced antioxidant capacity and enhanced oxidative damage in Keshan disease patients in vivo. There may be a certain relationship between oxidative stress disorder and myocardial damage of Keshan disease.     

肺癌胸腔镜手术患者的多学科加速康复外科护理

目的探讨多学科加速康复外科在肺癌电视胸腔镜手术(VATS)围手术期患者管理中的应用效果。方法将行VATS下肺癌根治术的121例患者按时间段分为对照组59例,行常规围手术期护理;62例为观察组,采用多学科加速康复外科进行术前术中及术后护理干预。结果观察组患者术后1h、6h、12h及24h的VAS评分显著低于对照组(均P0.01),术后肛门排气、下床活动、胸管留置及术后住院时间显著短于对照组(均P0.01),未发生肺不张及静脉血栓栓塞。结论多学科加速康复护理干预可有效减轻肺癌VATS术后患者疼痛,防范术后并发症,加速患者康复。     

腹部手术后近期发生肠梗阻54例临床分析

我们于1978年1月～1989年12月,腹部手术后第4～21天(?)近期出现的肠梗阻患者54例,现报告如下: 临床资料 1.性别与年龄:本组男40例,女14例。6～20岁14人,21～50岁28人、51～65岁8人,66～72岁4人。 2.梗阻与第一次手术的病种:胃十二指肠手术20例;肝脾破裂术后5例,胆囊穿孔手术3例,小肠手术12例(其中肠伤寒穿孔1例),结肠手术4例、阑尾炎术后8例,腹式输卵管结扎手术2例。 3.梗阻的种类及治则:本组单纯性粘连梗阻30