收费全文 | 119篇 |
免费 | 0篇 |
国内免费 | 3篇 |
儿科学 | 1篇 |
妇产科学 | 1篇 |
基础医学 | 16篇 |
口腔科学 | 1篇 |
临床医学 | 1篇 |
内科学 | 8篇 |
神经病学 | 64篇 |
特种医学 | 2篇 |
外科学 | 21篇 |
预防医学 | 2篇 |
眼科学 | 1篇 |
药学 | 3篇 |
肿瘤学 | 1篇 |
2023年 | 2篇 |
2022年 | 1篇 |
2020年 | 1篇 |
2019年 | 1篇 |
2018年 | 4篇 |
2016年 | 3篇 |
2015年 | 3篇 |
2014年 | 2篇 |
2013年 | 3篇 |
2012年 | 6篇 |
2011年 | 8篇 |
2010年 | 5篇 |
2009年 | 2篇 |
2008年 | 3篇 |
2007年 | 9篇 |
2006年 | 4篇 |
2005年 | 4篇 |
2004年 | 6篇 |
2003年 | 8篇 |
2002年 | 7篇 |
2001年 | 5篇 |
2000年 | 3篇 |
1999年 | 6篇 |
1998年 | 2篇 |
1995年 | 1篇 |
1992年 | 3篇 |
1991年 | 2篇 |
1990年 | 1篇 |
1989年 | 3篇 |
1988年 | 1篇 |
1987年 | 2篇 |
1986年 | 1篇 |
1985年 | 2篇 |
1984年 | 1篇 |
1983年 | 3篇 |
1981年 | 1篇 |
1980年 | 1篇 |
1978年 | 1篇 |
1967年 | 1篇 |
Methods: Spinal ischemia was induced by aortic occlusion lasting 10 min. After ischemia, spinal hypothermia was induced using a subcutaneous heat exchanger. Three studies were conducted. In the first study, the intrathecal temperature was decreased to 34, 30, or 27 [degree sign]C for 2 h beginning with initial reperfusion. In the second study, hypothermia (target intrathecal temperature 27 [degree sign]C) was initiated with reflow and maintained for 15 or 120 min. In the third study, the intrathecal temperature was decreased to 27 [degree sign]C for 2 h starting 5, 60, or 120 min after normothermic reperfusion. Animals survived for 2 or 3 days, at which time they were examined and perfusion fixed with 4% paraformaldehyde.
Results: Normothermic ischemia followed by normothermic reflow resulted in spastic paraplegia and spinal neuronal degeneration. Immediate postischemic hypothermia (27 [degree sign]C for 2 h) resulted in decreasing motor dysfunction. Incomplete protection was noted at 34 [degree sign]C. Fifteen minutes of immediate cooling (27 [degree sign]C) also provided significant protection. Delay of onset of post-reflow hypothermia (27 [degree sign]C) by 5 min or more failed to provide protection. Histopathologic analysis revealed temperature-dependent suppression of spinal neurodegeneration, with no effect of delayed cooling. 相似文献
Sports training and professional experience lead to changes in the human motor cortex and on the autonomic nervous system. This experimental study aims to investigate as sport activities can lead changes on the human motor cortex and on the autonomic nervous system (ANS), analyzing the Heart Rate changes and spontaneous Galvanic Skin Response (GSR) fluctuations in relation to the adaptive changes on the human motor cortex.
MethodsThree neurophysiologic parameters (rMT, MEP latency and MEP amplitude) were investigated using transcranial magnetic stimulation (TMS). TMS was applied to the primary motor cortex (M1) of 30 right-handed young karate athletes recruited. To evaluate ANS, HR (at rest and during exercise) and GSR (at rest and post-exercise) were measured. All data were matched with the records obtained by 30 non-athletes. All statistical analyses were performed using R.
ResultsStatistical significant differences were detected analyzing the data refers to neurophysiologic parameters. The HR and GSR values showed significant differences comparing athletes group with non-athletes group.
ConclusionsOur data suggest that there are significant differences analyzing the cortical excitability in athletes, compared to non-athletes. Furthermore, this data confirmed that the exercise training influences the parasympathetic tone, reducing HR. Moreover, a significant reduction in GSR parameters was reported in the athletes group, which is an indicator of stress level. The HR training helps to restore the autonomic balance and improves autonomic control that supports emotional regulation and performance coordination, so the training represents an advantage in reducing anxiety in athletes. Future studies could be used to investigate the differences of these adaptive changes respect to athletes’ level (for example, amateur vs professional).
相似文献