特发性室性心动过速射频消融治疗的临床研究

射频消融３１例特发性室性心动过速（ＩＶＴ）,对其定位、标测方法及复发原因进行探讨。采取激动和（或）起搏标测结合的方法进行标测和消融治疗。结果：右室ＩＶＴ１３例。起源于流出道部位１２例,其Ｉ导联以Ｑ波为主者偏前,ｒ波为主者偏后;Ｖ３导联的Ｒ／Ｓ＞１者在上部,Ｒ／Ｓ＜１者在下部。游离壁１例电轴左偏。起搏标测靶点心电图ＩＶＴ时与１２导联心电图一致,局部电位提前４２．０８±８．９１ｍｓ。１例术后复发。左室ＩＶＴ１８例。起源间隔者１４例,随着起源点从基底部向心尖部移行,电轴更右偏;游离壁４例电轴明显左偏。靶点局部电图均提前３２．１９±８．３６ｍｓ。左室间隔部ＩＶＴ记录到浦肯野纤维电位（ＰＰ）７例、异常电位（ＡＰ）４例;１例未记录到ＰＰ及ＡＰ且起搏标测１０导联心电图一致,术后复发。２例未诱发ＩＶＴ,均以起搏标测消融成功。左室游离壁起搏标测１２导联心电图一致３例,１１导联一致者１例且术后复发。３例４例次复发,１例（左室间隔ＩＶＴ）未再接受消融治疗,２例最终采用温控导管消融成功。结论：体表心电图对ＩＶＴ起源点定位有指导意义;激动标测与起搏标测相结合的方法更好。     

房室结慢径区域消融对迷走神经功能及心房颤动易感性的影响

Objective Atrioventricular node reentrant tachycardia (AVNRT) ablation may effect the vagal response,which is indicated by sinus tachycardia. On the other hand,atrial fibrillation (AF) ,which was found to be associated with vagal irmervation, often coexists with AVNRT. However,little is known about the im-pact of slow pathway ablation on local vagal innervation to atria. Methods In 11 dogs, bilateral cervical sympa-thovagal trunks were decentralized and metoprolol was given to block sympathetic effects. Linear lesion was per-formed from coronary sinus (CS) ostium to the middle area of Koch triangle. Atrial effective refractory period(ERP) ,vulnerability window (VW) of AF, and sinus rhythm cycle length (SCL) were measured at high fight atrium (HRA),low right atrium (LRA), distal (CSd) and proximal CS (CSp) at baseline with and without vagal stimulation before and after ablation. The histological study was also performed. Results (1) SCL during vagal stimulation remained unchanged before and after ablation(107±19)bpm vs (108±8) bpm (P > 0.05). (2) After ablation, ERP during vagal stimulation remained unchanged at HRA (55±34) ms vs (69 ±37) ms (P >0.05),and decreased slightly at CSd (42±32) ms vs (55±30) ms (P =0.08). However,at LRA and CSp,ERP was significantly decreased after ablation (19±21) ms vs (66±24) ms (P <0.001) ; and (7± 18) ms vs (46±24) ms (P < 0.001), respectively. (3) AF was difficult to be induced at baseline before and after ablation in all sites (VW close to 0). While during vagal stimulation, after ablation VW of AF significantly decreased at LRA (1±3) ms vs (49±36) ms (P < 0.005) and CSp (10±12) ms vs (45±34) ms (P < 0.05) ,decreased slightly at CSd after ablation (35±37) ms vs (57±28) ms (P =0.07) ,and remained un-changed at HRA (63±31) ms vs (63±25) ms (P > 0.05). (4) The altered architecture of individual gan-glia was histologically observed. Conclusions The decreased ERP shortening to vagal stimulation in CS and LRA induced by slow pathway ablation indicates that ablation in such area may result in the vagal dennervation in LRA and CS,thereby attenuating the susceptibility to vagal mediated AF. While unchanged SCL,ERP short-ening and VW to vagal stimulation in sinus node area and HRA indicate that slow pathway ablation did not change the vagal innervation to these sites.     

经左主动脉窦消融反复性单形室性心动过速一例

右心室流出道是反复性单形室性心动过速 (repetitivemonomorphic ventricular tachycardia,RMVT )的常见起源部位 ,其电生理特点及导管射频消融治疗已被大多数医师所熟知 ,但起源于左心室流出道的 RMVT报道较少 [1 - 3 ] 。本文报告 1例经左主动脉窦成功消融的 RMVT,并结合近几年文献对其电生理特点及射频消融的方法进行分析。　　资料和方法　患者女性 ,19岁。有阵发性心悸病史半年 ,心悸发生时伴有头晕、乏力、活动受限。临床检查 (包括超声心动图、X线三位胸片、核磁共振、冠状动脉造影 )无器质性心脏病的诊断依据。心电图及动态心…     

心脏骤停的抢救

心脏有节律的收缩和舒张是维持血液循环的动力,称为心脏的血泵功能。心脏骤停就是突然发生心脏血泵功能的丧失或停止,随之血液循环亦停止了,它是临床上最紧急的情况,若没有及时进行正确有效的抢救,迅速恢复心脏血泵机能,必然导致病人死亡。因此,在发生心脏骤停的现场,争分夺秒的进行抢救是最重要的环节。     

家族性良性房颤3例随访8年报告

1975年为转律收治一例年为27岁心房纤颤病人,各项检查未发现器质性心脏病其他证据,药物与电击复律均未能终止房颤恢复窦性心律,以良性房颤诊断出院,随后对其家族三代13人进行了体检及ECG检查,共发现房颤3例,随访8年,现简要报告如下。     

起搏导管引起自主心动心内膜摩擦音一例

患者男性,69岁。因反复晕厥、严重心动过缓于1989年10月3日入院。既往有高血压病史7年,心绞痛病史3年。近年来,数次突然意识丧失,大小便失禁约一分钟后缓解。查体:意识清楚。血压21.3/9.3kPa。颈静脉怒张。双肺底闻及湿罗音。心界向左扩大,心率31次/分,律齐。各瓣膜区无杂音。肝肋下3.0cm,下肢无水肿。心电图示窦缓、窦房阻滞、窦停搏、交界区逸搏心律,时而呈快速房颤。扇扫见左室、     

当今对持续性交接区反复性心动过速的认识

当今对持续性交接区反复性心动过速的认识持续性交接区反复性心动过速（ｐｅｒｍａｎｅｎｔｊｕｎｃｔｉｏｎａｌｒｅｃｉｐｒｏｃａｔｉｎｇｔａｃｈｙｃａｒｄｉａ，ＰＪＲＴ）是Ｃｏｕｍｅｌ于１９６７年首先报告的一种特殊的心动过速。早期工作认为ＰＪＲＴ是不典型...     

我用呼吸疗心悸

一个偶然的机会,一位经常心慌的患者,学会了“气气归脐”呼吸法,练习之后竟使心悸不药而愈。请看患者的方法介绍,还有心内科专家的点评。     

非阵发性房性心动过速伴Ⅱ度异位传出阻滞和文氏Ⅱ度房室阻滞

本文报告一例极少见的心电图。特点是一系列异位 P′—QRS—T,Ⅱ、Ⅲ、aVF、Ⅰ、Ⅴ_5导联 P′波倒置,aVR 导联 P′波直立,为逆行 P 波,P′—P′等于0.48s 到0.50s,P′波率为120次/min,P′—R 大于0.12s,等于0.14～0.16s,QRS 波形正常,符合非阵发性房性心动过速。长的异位 P′—P′间距等0.96～1.00s,恰好等于短 P′—P′间距的倍数,故存在Ⅱ度异位传出阻滞。同时可见 P′—R 间期逐渐延长,R—R 间期逐渐缩短,并随之出现一个长间期,所以有文氏Ⅱ度房室阻滞。还可以见到,介于窦性 P 波与房件 P′波之间形态的 P 波,为房性融合波。     

阵发性房颤患者大静脉肌袖与心房电连接特征的临床研究(摘要)

目的：总结阵发性房颤病人肺静脉和／或上腔静脉肌袖的电生理标测和导管射频消融电隔离的结果，评价国人大静脉肌袖和心房的电连接类型和特点。资料与方法：顽固性阵发性房颤患者45例，在环状标测电极指导下行大静脉肌袖的电位记录、分析以及对能标测到袖电位的大静脉进行开口部的点或段的消融电隔离治疗。根据窦律和心房起搏下的肌袖内环形电极标测到的袖电位的激动顺序以及有效放电对袖电位的影响，总结和分析袖房之间的电连接特点。结果：共标测和／或电隔离肌袖115根，其中肺静脉100根，上腔静脉15根。其中呈单束状电连接43根（38％），双束状电连接54根（46％），多束状电连接12根（10％），环状电连接3根，无电连接3根。结论：根据环状电极标测到的袖电位的激动顺序和对放电的反应，提示袖房之间电连接的类型多为单束状和双束状（84％），说明对于大多数肌袖并不需要行环状消融，而只要在袖房连接处行点或节段性消融即可达到完全袖房电隔离的结果。