CT在骨科中的应用

目的 :探讨 CT在骨骼肌肉系统疾病的应用价值。方法 :回顾性分析经病理证实或临床随访证实的 15 89例骨骼肌肉系统病变的 CT资料 ,并与临床进行对照。结果 :创伤性病变 15 6例 ,涉及颅面骨、脊柱、四肢、骨盆等部位 ,CT可显示复杂部位的细小骨折 ,以及邻近组织的伴随改变。骨关节病 13 79例 ,包括骨关节退行性变 ,无菌性坏死和骨纤维异常增殖症 ,CT可显示椎间盘病变的程度和部位 ,对无菌性坏死及骨纤维异常增殖症亦有独特的诊断价值。骨关节感染性病变 6例 ,肿瘤性病变 13例 ,软组织病变 3 5例 ,对上述 3类疾病 CT亦可提供明确的诊断信息。结论 :CT在骨关节系统具有十分明确的诊断价值 ,可发现常规 X线不能发现的早期表现和微小改变 ,有利于临床制订治疗方案 ,并可用于临床治疗的疗效观察     

64排螺旋CT对腰椎间盘病变的诊断价值

目的:探讨腰椎间盘病变在64排螺旋CT三维及多平面重建的影像学特征,提高腰椎间盘病变的诊断率。方法:对79例腰椎间盘病变部位行64排CT螺旋扫描(其中14例先行椎管造影),并进行三维重建、矢状重建及轴位重建。结果:对腰椎、骶椎共79例病变部位行矢状重建,其中腰间盘膨出31例,腰间盘突出44例,正常4例。64排螺旋CT三维重建的影像学特征为立体显示腰椎的整体形态;矢状重建的影像学特征为类似MR的图像,纵向显示脊椎的排列情况及椎管的整体形态,造影后矢状图像可以清晰地显示脊髓情况。结论:64排螺旋CT能提供三维立体图像及类似MR的腰椎图像,对腰椎间盘病变的诊断具有重要价值。     

64排螺旋CT靶重建技术在孤立性肺结节诊断中的应用

目的：探讨64排螺旋CT靶重建技术在孤立性肺结节诊断中的价值。方法：对常规64排CT肺部扫描发现的30例SPN患者进行靶重建,分析其影像特点,比较两种方法的诊断符合率。结果：30例靶重建显示小结节征8例、空泡征9例、钙化4例、分叶征16例、毛刺征12例、棘突征13例、胸膜凹陷征12例、血管集束征7例及20例增强病例显示密度变化均高于常规扫描;靶重建后诊断符合率为90%,也高于常规扫描的66.7%。结论：靶重建技术有助于提高孤立性肺结节（SPN）的诊断及鉴别诊断。     

The New Brunswick Health Council at a glance

This article presents an overview of the work of the New Brunswick Health Council (NBHC) since its creation by exploring its mandate and by presenting its different initiatives and publications as they relate to the NBHC's areas of work: population health, care experience, sustainability, and citizen engagement.     

爆裂性骨折所致眼球内陷的CT研究

目的:研究眼眶爆裂性骨折后,眼球内陷与眼眶容积扩大的关系并对眼球内陷进行早期预报。方法:28例单侧眼眶爆裂性骨折后1～350 d患者,分别测量双侧眼球位置和骨性眼眶容积值,计算患侧眼球内陷值(E)和骨性眼眶容积扩大值(V),应用pearson积矩法研究E、V相关性并建立直线回归方程。结果:28例患侧E、V呈正相关(r=0.88,P<0.001);15例≤24 d的患者直线回归方程为E=0.85 V-1.74(r=0.86,P<0.001),13例>24 d的患者直线回归方程为E=0.86 V-0.60(r=0.9,P<0.001)。结论:眼球内陷值与眼眶容积扩大值高度相关;1 mL眼眶容积的扩大,可引起大约0.9mm(0.85～0.86 mm)的眼球内陷。与伤后>24 d的相比,≤24 d的眼球有明显的抗拒内陷的趋势,>24 d之后,这种趋势逐渐消失,随之发生眼球内陷。     

无症状性脑梗塞的CT表现及病因分析

本文说明了无症状性脑梗塞的病灶大小、分布、特点及CT表现。主要病因是动脉粥样硬化、高血压、糖尿病。     

急性脑缺血的弥散和灌注磁共振成像

最近 ,磁共振成像 (MRI) ,特别是弥散加权成像 (DWI)和灌注加权成像 (PWI)的进展 ,已使临床医生具备了区分不可逆性脑梗死和潜在的可逆性缺血半暗带的能力。本文探讨了DWI和PWI的工作原理和应用。随着急性脑缺血溶栓和其他治疗的不断进展 ,在急性卒中的决策方面 ,神经影像学会随时起到越来越重要的作用。     

Porokeratosis Causing Change in Melanocytic Nevi

Large cell transformation of mycosis fungoides (MF) is an uncommon phenomenon. We present a case of CD30‐positive large cell transformation and discuss its possible pathophysiology. A 74 year‐old male with a 36‐year history of patch stage MF presented with a 3‐month history of right chest cellulitis that was refractory to IV antibiotic treatment. Skin biopsies from his thigh demonstrated a patchy dermal infiltrate of irregular and hyperchromatic lymphocytes and epidermotropism. The majority of the infiltrate was positive for CD4, CD3, CD2, and negative for CD7. Only 10% were positive for CD25 and CD8. Biopsies obtained from the ulcerated chest nodules showed a dermal infiltrate of large and pleomorphic lymphoid cells with prominent nucleoli. These large lymphoid cells were strongly positive for CD3, CD30, CD25, CD2 and UCHL‐1. Occasional cells were positive for CD4 and CD20. They were negative for ALK‐1, TIA‐1, CD7, CD8, and CD15. T‐gamma receptor gene rearrangement analyses by polymerase chain reaction demonstrated a clonal process with similar rearrangement patterns identified in the patch stage MF as well as in large cell transformation areas. Examinations of his peripheral blood and bone marrow were negative. The patient had tolerated one cycle of CHOP chemotherapy.