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Vivien M. Hsu Lorinda Chung Laura K. Hummers Fredrick Wigley Robert Simms Marcy Bolster Rick Silver Aryeh Fischer Monique E. Hinchcliff John Varga Avram Z. Goldberg Chris T. Derk Elena Schiopu Dinesh Khanna Lee S. Shapiro Robyn T. Domsic Thomas Medsger Maureen D. Mayes Daniel Furst Mary E. Csuka Jerry A. Molitor Firas Alkassab Virginia D. Steen 《Seminars in arthritis and rheumatism》2014
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Jason C. Jacob Penelope Armada Pavittarpaul Dhesi Firas Elbash Charles Primiano Elizabeth D. Estrada Paul D. Thompson 《The Canadian journal of cardiology》2012
Prader–Willi syndrome (PWS) is a syndrome characterized in babies by small birth weight, hypogonadism, flaccid muscle tone, and skeletal abnormalities, and in older children by intense food cravings leading to morbid obesity, hypoxemia, and right heart failure. To our knowledge, PWS has not been associated with coronary artery dissection. We report a 17-year-old woman with PWS who suffered an inferior myocardial infarction secondary to dissection of her right coronary artery. 相似文献
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Stephanie Tobin Firas Sedki Zarish Abbas Uri Shalev 《The European journal of neuroscience》2013,37(6):972-981
The neurotransmitter dopamine (DA) plays a critical role in both priming‐and cue‐induced reinstatement of extinguished drug‐seeking behavior, but its role in stress‐induced reinstatement is less clear. Our laboratory has recently demonstrated that systemic administration of the DA D1‐like receptor antagonist, SCH 23390, attenuates acute food deprivation (FD) stress‐induced reinstatement. The current study was designed to elucidate the brain regions critical to the effect of SCH 23390 on FD stress‐induced reinstatement. Rats were trained to press a lever to self‐administer heroin (0.1 mg/kg/inf) over a period of 10 days. Following training, heroin was removed leading to an extinction of lever pressing. Next, rats were tested for reinstatement twice, under extinction conditions: once following 21–48 h FD; and once under sated conditions. Prior to testing, SCH 23390 was administered into the nucleus accumbens (NAc) shell (0.0, 0.3, 0.6 μg/side), NAc core (0.0, 0.3, 0.6 μg/side), dorsomedial prefrontal cortex (dmPFC; 0.0, 0.2, 2.0 μg/side), ventromedial prefrontal cortex (vmPFC; 0.0, 2.0 μg/side) or basolateral amygdala (BLA; 0.0, 1.0, 2.0 μg/side). An attenuation of FD‐induced reinstatement of heroin seeking was seen in rats injected with SCH 23390 into the NAc shell, dmPFC or BLA, but not into the NAc core or the vmPFC. These findings support the hypothesis that DA transmission through the DA D1‐like receptors plays a critical role in stress‐induced reinstatement of heroin seeking. 相似文献
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