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排序方式: 共有2447条查询结果,搜索用时 46 毫秒
81.
Richard O. Phillips Michael Frimpong Fred S. Sarfo Birte Kretschmer Marcus Beissner Alexander Debrah Yaw Ampem-Amoako Kabiru M. Abass William Thompson Mabel Sarpong Duah Justice Abotsi Ohene Adjei Bernhard Fleischer Gisela Bretzel Mark Wansbrough-Jones Marc Jacobsen 《Emerging infectious diseases》2014,20(6):1000-1003
During August 2010–December 2012, we conducted a study of patients in Ghana who had Buruli ulcer, caused by Mycobacterium ulcerans, and found that 23% were co-infected with Mansonella perstans nematodes; 13% of controls also had M. perstans infection. M. perstans co-infection should be considered in the diagnosis and treatment of Buruli ulcer. 相似文献
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Barboi Alexandru Gibbons Christopher H. Bennaroch Eduardo E. Biaggioni Italo Chapleau Mark W. Chelimsky Gisela Chelimsky Thomas Cheshire William P. Claydon Victoria E. Freeman Roy Goldstein David S. Joyner Michael J. Kaufmann Horacio Low Phillip A. Norcliffe-Kaufmann Lucy Robertson David Shibao Cyndya A. Singer Wolfgang Snapper Howard Vernino Steven Raj Satish R. 《Clinical autonomic research》2020,30(2):183-184
Clinical Autonomic Research - 相似文献
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Bodil Nielsen Gisela SjØgaard Flemming Bonde-Petersen 《European journal of applied physiology》1984,53(1):63-70
Summary During prolonged heavy exercise a gradual upward drift in heart rate (HR) is seen after the first 10 min of exercise. This secondary rise might be caused by a reduction in stroke volume due to reduced filling of the heart, which is dependent upon both hemodynamic pressure and blood volume. Swimming and bicycling differ with respect to hydrostatic pressure and to water loss, due to sweating. Five subjects were studied during 90 min of bicycle exercise, and swimming the leg kick of free style. The horizontal position during swimming resulted in a larger cardiac output and stroke volume. After the initial rise in heart rate the secondary rise followed parallel courses in the two situations. The rises were positively related to the measured increments in plasma catecholamine concentrations, which continued to increase as exercise progresssed. The secondary rise in HR could not be explained by changes in plasma volume or in water balance, nor by changes in plasma [K]. The plasma volume decreased 5–6% (225–250 ml) within the first 5 to 10 min of exercise both in bicycling and swimming, but thereafter remained virtually unchanged. The sweat loss during bicycling was four times greater than during swimming; but during swimming the hydrostatic conditions induced a diuresis, so that the total water loss was only 25% less than during bicycling. 相似文献
88.
Daniel P. Potaczek Sebastian D. Unger Nan Zhang Styliani Taka Sven Michel Nesibe Akdağ Feng Lan Markus Helfer Christoph Hudemann Markus Eickmann Chrysanthi Skevaki Spyridon Megremis Anne Sadewasser Bilal Alashkar Alhamwe Fahd Alhamdan Mübeccel Akdis Michael R. Edwards Sebastian L. Johnston Harald Renz 《The Journal of allergy and clinical immunology》2019,143(4):1403-1415
89.
Timo Siepmann Manfred Tesch Florian Krause Ben Min-Woo Illigens Gisela Stoltenburg-Didinger 《Annals of diagnostic pathology》2013,17(2):183-186
Polymyositis (PM) with cytochrome C oxidase negative fibers also referred to as PM with mitochondrial pathology (PM-Mito) is characterized by the symptoms of inclusion body myositis (IBM) and by the myopathological findings of PM except for an increase of muscle fibers with insufficient mitochondrial cytochrome C oxidase activity. Few PM-Mito cases are published; mitochondrial ultrastructure has not been studied in these patients. We report 2 PM-Mito patients with later onset than usually seen in IBM and poor responsiveness to glucocorticoids. Electron microscopy of muscle fibers showed irregular mitochondrial ultrastructure. Sjögren syndrome related antinuclear antibodies (Anti-Ro and Anti-La) were found in one of the two patients but the typical clinical symptoms of Sjögren syndrome such as xerostomia and keratoconjunctivitis were absent in this patient. Taken together, our observations, viewed in conjunction with the current literature, suggest that PM-Mito is an underdiagnosed disease with a multifactorial pathogenesis that should be elucidated in further studies. We want to encourage clinicians and pathologists to consider the possibility of PM-Mito in patients with atypical PM or sIBM. 相似文献
90.
Israel Castillo-Juárez Violeta González Héctor Jaime-Aguilar Gisela Martínez Edelmira Linares Robert Bye Irma Romero 《Journal of ethnopharmacology》2009