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REMATCH Investigators William L. Holman MD Soon J. Park MD James W. Long MD PhD Alan Weinberg MS Lopa Gupta RD MPH Anita R. Tierney MPH Robert M. Adamson MD John D. Watson MD Edward P. Raines MD JD Gregory S. Couper MD Francis D. Pagani MD PhD Nelson A. Burton MD Leslie W. Miller MD Yoshifumi Naka MD PhD 《The Journal of heart and lung transplantation》2004,23(12):E84-1365
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H van der Berg K Wage JD Burggraaf M Peters 《Acta paediatrica (Oslo, Norway : 1992)》1997,86(7):778-780
Abstract In an infant with skeletal anomalies and haemolytic disease, intestinal perforation was caused by necrosis of an as yet undetected B-cell lymphoma. Severe combined immunodeficiency with short-limbed skeletal dysplasia was diagnosed. This is the first published report of a patient with this syndrome in combination with haemolytic disease and B-cell-lymphoma. 相似文献
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Gap junction intercellular communication (GJIC) has been measured in cell
lines that represent different stages of chemically induced mouse skin
carcinogenesis. No significant difference in GJIC, as measured by dye
spread, was found in cultures of normal keratinocyte, papilloma or squamous
carcinoma cell lines. There was no correlation, in this system, between the
presence of a mutant Ha-ras gene and down- regulation of communication.
There was, however, a marked decrease in GJIC (80-90%) on progression from
squamous to spindle carcinoma cells. Measurement of GJIC in somatic cell
hybrids shows that the genetic defect responsible for this down-regulation
is recessive and is common to two independently isolated spindle cell
lines. No abnormalities were found in the spindle cells in expression of
connexin 43, a cell component involved in gap junction formation and
permeability. However, expression of E-cadherin, a cell-cell adhesion
molecule implicated in the process of gap junction formation, was missing
in the spindle carcinoma cells. Introduction of an E-cadherin cDNA into the
spindle cells partially restored junctional communication without causing
any noticeable alterations in cell morphology. During the study a non-
tumourigenic keratinocyte line, a sub-clone of a normal keratinocyte line,
was also found to have a low level of GJIC. However, the defect in this
line was shown, by genetic complementation in somatic cell hybrids, to be
different from that in the spindle carcinoma cell lines. Consistent with
these data, analysis by immunofluorescence shows an abnormal distribution
of connexin 43 in these cells.
相似文献
180.
Exposure and peritraumatic response as predictors of posttraumatic stress in children following the 1995 Oklahoma City bombing 总被引:1,自引:0,他引:1
Drs. Betty Pfefferbaum MD JD Mr. Debby E. Doughty Chandrashekar Reddy Drs. Nilam Patel Dr. Robin H. Gurwitch Drs. Sara Jo Nixon Mr. Rick D. Tivis 《Journal of urban health》2002,79(3):354-363
Studies have demonstrated a positive relationship between exposure and posttraumatic stress, but one's subjective appraisal
of danger and threat at the time of exposure may be a better predictor of posttraumatic stress than more objective measures
of exposure. We examined the role of peritraumatic response in posttraumatic stress reactions in over 2,000 middle school
children 7 weeks after the 1995 Oklahoma City, Oklahoma, bombing. While many children reported hearing and feeling the blast
and knowing direct victims, most were in school at the time of the explosion and therefore were not in direct physical proximity
to the incident. Physical, interpersonal, and television exposure accounted for 12% of the total variance in our measure of
posttraumatic stress when peritraumatic response was ignored. Peritraumatic response and television exposure accounted for
25% of the total variance, and physical and interpersonal exposure were not significant in this context. These findings suggest
the importance of peritraumatic response in children's reactions to terrorism. These carly responses can be used to help determine
which children may experience difficulty over time. 相似文献