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41.
I. Costero C. Neuhaus Hoffmann Plehn V. E. Mertens Hermann Groß Werthemann Maximilian Hirsch H. Brach Feles E. Törö Mildenberger Hadda Janke Sponholz A. Rosenburg E. Zurhelle Collier 《Journal of cancer research and clinical oncology》1931,34(6):219-235
Ohne Zusammenfassung 相似文献
42.
Orzechowski Hartmann A. Fröhlich Haagen R. Henneberg Werthemann Krauspe Böhmig Collier Fuchs Langendorff Rother Sponholz F. E. Lehmann 《Journal of cancer research and clinical oncology》1936,44(3-4):184-192
Ohne Zusammenfassung 相似文献
43.
Hartmann Sponholz Haagen Krah A. Th. Czaja 《Journal of cancer research and clinical oncology》1936,44(3-4):173-178
Ohne Zusammenfassung 相似文献
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Knake Wassermann Stöhr Jr. Campbell Hans Ehrlich H. Brach Sponholz Fritz Schmincke Plehn Robert Meyer A. Rosenburg Peter Feles Rusch Cohrs 《Journal of cancer research and clinical oncology》1930,32(3):5-14
Ohne Zusammenfassung 相似文献
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Endothelial nitric oxide synthase mediates protective effects of hypoxic preconditioning in lungs 总被引:2,自引:0,他引:2
Kaminski A Kasch C Zhang L Kumar S Sponholz C Choi YH Ma N Liebold A Ladilov Y Steinhoff G Stamm C 《Respiratory physiology & neurobiology》2007,155(3):280-285
To elucidate the protective mechanism of whole-body hypoxic preconditioning (WHPC) on pulmonary ischemia-reperfusion injury focussing on nitric oxide synthases (NOS), mice were placed in a hypoxic chamber (FIO(2)=0.1) for 4h followed by 12h of normoxia. Then, pulmonary ischemia for 1h followed by 5h of reperfusion was performed by clamping the left hilum in vivo (I/R). WHPC protected WT mice from pulmonary leukocyte infiltration as assessed by myeloperoxidase (MPO) activity, associated with a mild further increase in endothelial permeability (Evans Blue extravasation). When all NOS isoforms were inhibited during WHPC by L-NAME, mortality and MPO activity after I/R markedly increased. To determine the responsible NOS isoform, quantitative RT-PCR was performed for eNOS and iNOS mRNA, showing that only eNOS was upregulated in response to WHPC. While eNOS total protein expression remained unchanged, the amount of phosphorylated eNOS also increased. The WHPC/IR experiments were then repeated with eNOS knockout mice. Here, we found that the protective effect of WHPC on pulmonary leukocyte sequestration was abrogated, and endothelial leakage was further exacerbated. We conclude that WHPC limits neutrophil sequestration via an eNOS-dependent mechanism, and that eNOS helps preserve endothelial permeability during hypoxia and I/R. 相似文献
49.
André Nathan Costa Edson Marchiori Gil Benard Mariana Sponholz Araújo Bruno Guedes Baldi Ronaldo Adib Kairalla Carlos Roberto Ribeiro Carvalho 《Jornal brasileiro de pneumologia》2013,39(3):368-372
On HRCT scans, lung cysts are characterized by rounded areas of low attenuation in the lung parenchyma and a well-defined interface with the normal adjacent lung. The most common cystic lung diseases are lymphangioleiomyomatosis, Langerhans cell histiocytosis, and lymphocytic interstitial pneumonia. In a retrospective analysis of the HRCT findings in 50 patients diagnosed with chronic paracoccidioidomycosis, we found lung cysts in 5 cases (10%), indicating that patients with paracoccidioidomycosis can present with lung cysts on HRCT scans. Therefore, paracoccidioidomycosis should be included in the differential diagnosis of cystic lung diseases. 相似文献
50.
S. Sponholz S. Bölükbas S. Oguzhan M. Schirren Prof. Dr. J. Schirren 《Der Onkologe》2014,20(8):740-745