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排序方式: 共有6192条查询结果,搜索用时 31 毫秒
231.
Stephan Sachs PhD Lili Niu PhD Philipp Geyer PhD Sigrid Jall PhD Maximilian Kleinert PhD Annette Feuchtinger PhD Kerstin Stemmer PhD Markus Brielmeier DVM Brian Finan PhD Richard D. DiMarchi PhD Matthias H. Tschöp MD Nicolai Wewer Albrechtsen PhD Matthias Mann PhD Timo D. Müller PhD Susanna M. Hofmann MD 《Diabetes, obesity & metabolism》2021,23(1):195-207
232.
Krzysztof Milewski MD PhD Anna Bryła-Wojciechowska MS Piotr P. Buszman MD PhD Katarzyna Jelonek MS PhD Mateusz Kachel MD Paweł Gąsior MD PhD Agata Krauze MS PhD Aleksandra Błachut MD Monika Musiał-Kulik MS PhD Armando Tellez MD Serge D. Rousselle DVM R. Stefan Kiesz MD PhD Janusz Kasperczyk MS PhD Paweł E. Buszman MD PhD 《Catheterization and cardiovascular interventions》2021,98(5):914-922
233.
Vaibhav Bagaria Raju Vaishya 《Diabetes & Metabolic Syndrome: Clinical Research & Reviews》2021,15(4):102137
As COVID 19 continues to over burden the healthcare system globally, the scientists are relentlessly pursuing research and publishing copious data on relevant managements strategies for the infection. This short communication has attempted to simplify the available information on the subject in a manner that is easy to understand and implement in clinical setting. COVID 19 is not a single disease but a spectrum and should be classified based on clinical, radiological and laboratory parameters. A simple yet powerful way is to classify COVID 19 as COVIN – COVID Infection but no disease; COVIRI – COVID infection with predominant respiratory symptoms; COVIDI - COVID infection leading to an abnormal immune response and COVID S- referring to the sequalae of an acute COVID Infection. A clinical subtype specific approach may result in easier communication between healthcare providers which in turn may improve patient outcomes by providing targeted therapy. 相似文献
234.
Azmi Lubna Shukla Ila Gupta Shyam Sundar Yadav Narayan Prasad Kant Padam Rao Ch. V. 《Proceedings of the National Academy of Sciences, India. Section B.》2019,89(3):1039-1045
Proceedings of the National Academy of Sciences, India Section B: Biological Sciences - Chronic gastric ulcer being the most prevalent gastrointestinal disorder is considered a major cause of... 相似文献
235.
Brains of macaques inoculated with macrophage-tropic, neurovirulent virus 7F, with lymphocyte-tropic SIV mac239, or with dual-tropic SIVmac239/1yE, were examined for microglial activation, astrocyte activation, apoptosis and neuron loss. The brain one animal inoculated with neurovirulent virus 7f showed massive microglial activation as assessed by expression of the major histo-compatibility complex class II (MHC-II). In this animal very numerous, large microglial nodules expressing MHC-II were concentrated in the basal pons and internal capsule. These microglial nodules contained cells undergoing apoptosis detected by in situ end labeling of fragmented DNA. In this animal, neuron loss was apparent near the microglial nodules. In the animals inoculated with SIVmac239 or SIVmac239/17E, pathologic changes such as perivascular cuffing and formation of microglial nodules were absent. However, increased expression of MHC-11 by microglial cells was also concentrated in white matter of the basal pons, midbrain and internal capsule. These results indicate the microglial activation in SIV-infected macaques follows a ventral to dorsal gradient regardless of viral tropism. These results also show that the type and severity of neuropathological changes in SIV-infected macaques is highly dependent on the tropism of the inoculated virus. 相似文献
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237.
Mechanical bases of frequency tuning and neural excitation at the base of the cochlea: comparison of basilar-membrane vibrations and auditory-nerve-fiber responses in chinchilla 下载免费PDF全文
Ruggero MA Narayan SS Temchin AN Recio A 《Proceedings of the National Academy of Sciences of the United States of America》2000,97(22):11744-11750
We review the mechanical origin of auditory-nerve excitation, focusing on comparisons of the magnitudes and phases of basilar-membrane (BM) vibrations and auditory-nerve fiber responses to tones at a basal site of the chinchilla cochlea with characteristic frequency approximately 9 kHz located 3.5 mm from the oval window. At this location, characteristic frequency thresholds of fibers with high spontaneous activity correspond to magnitudes of BM displacement or velocity in the order of 1 nm or 50 microm/s. Over a wide range of stimulus frequencies, neural thresholds are not determined solely by BM displacement but rather by a function of both displacement and velocity. Near-threshold, auditory-nerve responses to low-frequency tones are synchronous with peak BM velocity toward scala tympani but at 80-90 dB sound pressure level (in decibels relative to 20 microPascals) and at 100-110 dB sound pressure level responses undergo two large phase shifts approaching 180 degrees. These drastic phase changes have no counterparts in BM vibrations. Thus, although at threshold levels the encoding of BM vibrations into spike trains appears to involve only relatively minor signal transformations, the polarity of auditory-nerve responses does not conform with traditional views of how BM vibrations are transmitted to the inner hair cells. The response polarity at threshold levels, as well as the intensity-dependent phase changes, apparently reflect micromechanical interactions between the organ of Corti, the tectorial membrane and the subtectorial fluid, and/or electrical and synaptic processes at the inner hair cells. 相似文献
238.
D P Sharma M Anderson M C Zink R J Adams A D Donnenberg J E Clements O Narayan 《The Journal of infectious diseases》1992,166(4):738-746
The simian immunodeficiency virus, SIVmac, causes disease affecting multiple organ systems in macaques similar to human immunodeficiency virus infection in humans. Molecularly cloned SIVmac with a strong lymphocyte tropism was used in pathogenesis experiments to correlate viral cell tropism with disease. In 5 animals, exhaustive analyses on viruses from tissues and identification of infected precursor cells were done at multiple times during infection to ensure the virus had not mutated into a macrophage-tropic variant. Viral replication was measured by infectivity, infectious center assays, and in situ hybridization. Lymphocytes produced most virus in tissues, indicating the virus maintained its cell tropism in vivo. Lymphocytes in bone marrow were latently infected and those in the spleen and lymph nodes were productively infected. The virus failed to replicate in the brain after intracerebral inoculation. SIVmac that maintained a strong tropism for lymphocytes and a corresponding poor tropism for macrophages can cause persistent infection and AIDS but not other diseases such as primary pneumonia and encephalitis in rhesus macaques. 相似文献
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Stephen J. Thomas PhD ATC Katherine E. Reuther BS Jennica J. Tucker BS Joseph J. Sarver PhD Sarah M. Yannascoli MD Adam C. Caro DVM Pramod B. Voleti MD Sarah I. Rooney MSE David L. Glaser MD Louis J. Soslowsky PhD 《Clinical orthopaedics and related research》2014,472(8):2404-2412