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Xin Zhang MD Xiao‐Wei Liu MD PhD Xiao‐Yan Gu MD PhD Jian‐Cheng Han MD PhD Xiao‐Yan Hao MD Yu‐Wei Fu MD Yi‐Hua He MD PhD 《Echocardiography (Mount Kisco, N.Y.)》2018,35(4):563-565
Berry syndrome is a rare congenital cardiac malformation. We describe 4 cases of Berry syndrome diagnosed by fetal echocardiography. Based on our experience, the three‐vessel view is important for diagnosing the aortopulmonary window and aortic origin of the right pulmonary artery. Furthermore, the true cross‐sectional and sagittal views obtained by continuously scanning from the three‐vessel‐trachea view to the long‐axis view of the aortic arch are required to image the interruption or coarctation of the aortic arch. An early and accurate prenatal diagnosis of Berry syndrome is feasible and helps to improve patient outcomes. 相似文献
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Dulaglutide decreases plasma aminotransferases in people with Type 2 diabetes in a pattern consistent with liver fat reduction: a post hoc analysis of the AWARD programme 下载免费PDF全文
K. Cusi N. Sattar L.‐E. García‐Pérez I. Pavo M. Yu K. E. Robertson C. A. Karanikas A. Haupt 《Diabetic medicine》2018,35(10):1434-1439
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Edmund Kenneth Kerut MD Philip G. Kogos MD Jason H. Anderson MD Michael Turner MD Robert Ascuitto MD Nancy Ross‐Ascuitto MD Sameh M. Said MD 《Echocardiography (Mount Kisco, N.Y.)》2018,35(7):1045-1048
A healthy 20‐year‐old male presented with aborted sudden death. Both thoracic echocardiography and cardiac computed tomographic angiography demonstrated the characteristic findings of ALCAPA noting a dilated and tortuous right coronary artery, dilated coronary collateral arteries within the interventricular septum or along the surface of the heart, and visualization of the left coronary artery origin from the posterior aspect of the main pulmonary artery. Both imaging modalities demonstrated all three characteristic findings of this rare coronary anomaly and served as complementary imaging studies for surgical correction. 相似文献
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Transesophageal echocardiography for the assessment of left atrial appendage thrombus: Study of the additional value of systematic real time 3D imaging after regular 2D evaluation 下载免费PDF全文
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Huayqui Volt José A. García Carolina Doerrier María E. Díaz‐Casado Ana Guerra‐Librero Luis C. López Germaine Escames Jesús A. Tresguerres Darío Acuña‐Castroviejo 《Journal of pineal research》2016,60(2):193-205
The connection between the innate immune system, clock genes, and mitochondrial bioenergetics was analyzed during aging and sepsis in mouse heart. Our results suggest that the sole NF‐κB activation does not explain the inflammatory process underlying aging; the former also triggers the NLRP3 inflammasome that enhances caspase‐1‐dependent maturation of IL‐1β. In this way, aged mice enter into a vicious cycle as IL‐1β further activates the NF‐κB/NLRP3 inflammasome link. The origin of NF‐κB activation was related to the age‐dependent Bmal1/Clock/RORα/Rev‐Erbα loop disruption, which lowers NAD+ levels, reducing the SIRT1 deacetylase ability to inactivate NF‐κB. Consequently, NF‐κB binding to DNA increases, raising the formation of proinflammatory mediators and inducing mitochondrial impairment. The cycle is then closed with the subsequent NLRP3 inflammasome activation. This paired contribution of the innate immune pathways serves as a catalyst to magnify the response to sepsis in aged compared with young mice. Melatonin administration blunted the septic response, reducing inflammation and oxidative stress, and enhancing mitochondrial function at the levels of nonseptic aged mice, but it did not counteract the age‐related inflammation. Together, our results suggest that, although with different strengths, chronoinflammaging constitutes the biochemical substrate of aging and sepsis, and identifies the NLRP3 inflammasome as a new molecular target for melatonin, providing a rationale for its use in NLRP3‐dependent diseases. 相似文献
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